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tenecteplase is cleared from circulation by binding to specific receptors in the liver followed by catabolism to small peptides.
tenecteplase går fri af cirkulationen ved at binde sig til specifikke receptorer i leveren efterfulgt af nedbrydning til små peptider.
no direct effect of hepatic function on the pharmacokinetics of reslizumab is expected because antibodies are principally cleared by catabolism.
det forventes ikke, at leverfunktionen har nogen direkte indvirkning på reslizumabs farmakokinetik, da antistoffer primært udskilles via katabolisme.
the majority of igg elimination occurs via intracellular catabolism, following fluid-phase or receptor mediated endocytosis.
hovedparten af igg-elimination finder sted via intracellulær katabolisme efter væskefase eller receptormedieret endocytose.
as an igg1 monoclonal antibody, daclizumab is expected to undergo catabolism to peptides and amino acids in the same manner as endogenous igg.
som et igg1 monoklonalt antistof forventes daclizumab at blive kataboliseret til peptider og aminosyrer på samme måde som endogent igg.
mucopolysaccharide storage disorders are caused by the deficiency of specific lysosomal enzymes required for the catabolism of glycosaminoglycans (gags).
mucopolysaccharidose skyldes mangel på specifikke lysosomale enzymer, der er nødvendige i kataboliseringen af glycosaminoglycans (gags).
by inhibiting the normal catabolism of tyrosine in patients with ht-1, nitisinone prevents the accumulation of the toxic intermediates maleylacetoacetate and fumarylacetoacetate.
ved at hæmme den normale katabolisme af tyrosin hos patienter med ht- 1, forhindrer nitisinon akkumuleringen af de toksiske mellemprodukter maleylacetoacetat og fumarylacetoacetat.
mucopolysaccharidoses comprises a group of lysosomal storage disorders caused by the deficiency of specific lysosomal enzymes required for the catabolism of glycosaminoglycans (gag).
mukopolysakkaridoser består af en gruppe af lysosomale ophobningssygdomme forårsaget af mangel på specifikke lysosomale enzymer, der er nødvendige for katabolisme af glykosaminoglykaner (gag).
this results in an increase in the number of ldl-receptors on cell surfaces and enhanced receptor-mediated catabolism and clearance of circulating ldl-cholesterol.
dette resulterer i en stigning i antallet af ldl-receptorer på celleoverfladerne samt en forstærket receptor-medieret katabolisme og clearance af ldl-kolesterol i blodet.
within muscle tissue this includes increasing glycogen, fatty acid, glycerol and protein synthesis and amino acid uptake, while decreasing glycogenolysis, gluconeogenesis, ketogenesis, lipolysis, protein catabolism and amino acid output.
i muskelvæv øges glykogen-, fedtsyre-, glycerol- og proteinsyntesen samt aminosyreoptagelsen, mens glykogenolysen, glukoneogenesen, ketogenesen, lipolysen, proteinkatabolismen og aminosyreudskillelsen falder.