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neutralisation of factor xa interrupts the blood coagulation cascade and inhibits both thrombin formation and thrombus development.
hæmning af faktor xa standser koagulationsprocessen, hvorved trombinsyntese og trombedannelse reduceres.
pci and risk of guiding catheter thrombus in stemi patients undergoing primary pci, the use of fondaparinux prior to and during pci is not recommended.
13 pci og risiko for ledekateter-trombose.stemi-patienter, som gennemgår primær pci, bør ikke behandles med fondaparinux før og under pci-proceduren.
anaphylactic reaction, anaphylactic shock including fatal shock, hypersensitivity, urticaria thrombus including fatal thrombus, haematoma intracranial haemorrhage
ukendt hyppighed anafylaktisk reaktion/shock, inklusive fatalt shock, hypersensitivitet, urticaria trombose inklusive fatal trombose, hæmatom intrakraniel blødning
a non-dose related incidence of thrombus/emboli in the lung was seen in the 1 month repeated dose study in the monkey.
en ikke dosisrelateret forekomst af blodprop i lungen (lungeemboli) blev konstateret i 1 måneders studiet med gentagne doser til aber.
when a blood vessel is damaged, for example if it is cut, platelets clump together to help form a blood clot (thrombus).
når en blodåre bliver beskadiget, for eksempel hvis der skæres i den, klumper blodpladerne sammen for at hjælpe til med at danne en klump af blod, også kaldet en blodprop.
since thrombin (serine protease) enables the conversion of fibrinogen into fibrin during the coagulation cascade, its inhibition prevents the development of thrombus.
da trombin (serinprotease) fremmer ændringen af fibrinogen til fibrin i koagulationskaskaden, vil en hæmning af trombin forhindre udviklingen af tromber.
in subjects undergoing primary pci the incidence of adjudicated guiding catheter thrombus was 1.2% vs 0% in fondaparinux vs. control subjects, respectively.
hyppigheden af trombose, vurderet som værende kateterrelateret, var for patienter, der fik foretaget primær pci, henholdsvis 1,2 % for patienter i fondaparinuxgruppen og 0 % i kontrolgruppen.
it binds to the fibrin component of the thrombus (blood clot) and selectively converts thrombus-bound plasminogen to plasmin, which degrades the fibrin matrix of the thrombus.
det binder sig til fibrinkomponenten af tromben (størknet blod) og konverterer selektivt trombebundet plasminogen til plasmin, som
efficacy across subgroups, including age, gender, body mass index (bmi), renal function, extent of index pe, location of dvt thrombus, and prior parenteral heparin use was generally consistent.
virkningen på tværs af undergrupper, herunder alder, køn, body mass index (bmi), nyrefunktion, omfang af le-indeks, placering af dvt-trombose og tidligere parenteral heparin, var generelt ens.