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sirolimus inhibits t cell activation induced by most stimuli, by blocking calcium dependent and calcium independent intracellular signal transduction.
11 sirolimus jinibixxi l- attivazzjoni taċ- ċelluli t li s- soltu sseħħ permezz ta ’ ħafna stimuli, billi dan jimblokka t- transduzzjoni ta ’ messaġġi intraċellulari kemm dawk li huma u li mhumiex dependenti mill- kalċju.
after epo binds to its cell surface receptor, it activates signal transduction pathways that interfere with apoptosis and stimulates erythroid cell proliferation.
wara li epo jeħel mar-riċettur tal-wiċċ taċ-ċellula tiegħu, hu jattiva rotot tat-transduzzjoni tas-sinjal li jinterferixxu mal-apoptosi u jistimulaw il-proliferazzjoni taċ-ċelluli eritrojdi.
ticagrelor does not interact with the adp binding site itself, but interacts with platelet p2y12 adp-receptor to prevent signal transduction.
ticagrelor ma jinteraġixxix mas-sit innifsu fejn jeħel l-adp, iżda jinteraġixxi mar-riċettur tal-p2y12 adp tal- plejtlets biex jevita li s-sinjal jibqa għaddej.
genes related to transfer and mobilization function (e.g. with respect to conjugation, transduction or chromosomal integration);
ġeni li jirrigwardaw funzjoni ta' trasferiment u mobilizzazzjoni (eż. rigward konjugazzjoni, trasduzzjoni jew integrazzjoni kromożomali);
pegvisomant binds to growth hormone receptors on cell surfaces, where it blocks growth hormone binding, and thus interferes with intracellular growth hormone signal transduction.
pegvisomant jeħel mar-riċetturi tal-ormoni tat-tkabbir fuq il-wiċċ taċ-ċelluli, fejn jimblokka t-tqabbid tal-ormoni tat-tkabbir u b’hekk ifixkel is-sinjal trasduzzjonali fl-ormoni tat-tkabbir intraċellulari.
sirolimus inhibits t-cell activation induced by most stimuli, by blocking calcium-dependent and calcium-independent intracellular signal transduction.
sirolimus jinibixxi l-attivazzjoni taċ-ċelluli t ikkawżata mill-biċċa l-kbira tal-istimuli, billi dan jimblokka t-transduzzjoni ta’ messaġġi intraċellulari kemm dawk li huma u li mhumiex dipendenti mill-kalċju.
ed dependent signal transduction pathways in t cells, thereby preventing the transcription and synthesis of il-2, il-3, il-4, il-5 and other cytokines such as gm-csf, tnf-α and ifn-γ.
t za jinibixxi ġewwa t- cells sinjali tat- transduzzjoni, li jiddependu fuq il- kalċju, u b' hekk tiġi mwaqqfa t - transkrizzjoni u s- sinteżi ta 'il- 2, il- 3, il- 4, il- 5, u ta' ċitokini oħra bħal gm- csf, tnf- α u ifn- γ.