Results for glucuronidation translation from Maltese to English

glucuronidation ta’ lamotrigine
bedaquiline:

passaġġi metaboliċi oħrajn jinkludu glucuronidation, methylation u reduzzjoni.
other metabolic pathways include glucuronidation, methylation and reduction.

perċentwali żgħir ta’ vitamina d3 issirilha glucuronidation qabel l-eliminazzjoni.
a small percentage of vitamin d3 undergoes glucuronidation prior to elimination.

n-methylation, n-glucuronidation u ossidazzjoni fl-imidazole ring ġew osservati wkoll.
n-methylation, n-glucuronidation and oxidation in the imidazole ring have also been observed.

mogħdijiet sekondarji ta’ bijotrasformazzjoni li jinvolvu prodotti primarji ta’ bijotrasformazzjoni inkludew glucuronidation u dehydrogenation.
secondary biotransformation pathways involving the primary biotransformation products included glucuronidation and dehydrogenation.

metabolit o-glucuronidation ta’ topotecan u n-desmethyl topotecan ġew identifikati fl-awrina.
an o-glucuronidation metabolite of topotecan and n-desmethyl topotecan has been identified in the urine.

metaboliti ta’ o-glucuronidation ta’ topotecan u n-desmethyl topotecan ġew identifikati fl-awrina.
an o-glucuronidation metabolite of topotecan and n-desmethyl topotecan has been identified in the urine.

in-vitro, glivec jinibixxi paracetamol o-glucuronidation b’valur ki ta’ 58.5 mikromol/l.
in vitro, glivec inhibits paracetamol o-glucuronidation with ki value of 58.5 micromol/l.

idelalisib huwa metabolizzat primarjament permezz ta’ aldehyde oxidase, u fuq livell inqas permezz ta’ cyp3a u glucuronidation (ugt1a4).
idelalisib is metabolised primarily via aldehyde oxidase, and to a lesser extent via cyp3a and glucuronidation (ugt1a4).

is-sustanza mill-metaboliżmu ta’ o-glucuronidation ta’ topotecan u desmethyl topotecan ġew identifikati fl-awrina.
an o-glucuronidation metabolite of topotecan and n-desmethyl topotecan has been identified in the urine.

Ġew identifikati ħames passaġġi minn fejn lasofoxifene jiġi mmetabolizzat: ditossifikazzjoni diretta mill- fwied fejn jinqabad il-glucuronic acid biex jitneħħa mill-ġisem; ditossifikazzjoni diretta mill-fwied fejn jinqabad il-kubrit biex jitneħħa mill-ġisem;–idrossilazzjoni mal-parti phenyl tertaline tal-molekula (b’metaboliżmu successiv konjugattiv ta’ intermedjarji ta’ catechol bil- proċess ta’ methylation u glucuronidation); ossidazzjoni fiċ-ċirku pyrrolidine; u phenyl idrossilazzjoni.
five metabolic pathways of lasofoxifene have been identified: direct glucuronidation; direct sulfation; hydroxylation at the phenyl tetraline moiety (with subsequent conjugative metabolism of the catechol intermediates by methylation and glucuronidation); oxidation at the pyrrolidine ring; and phenyl hydroxylation.