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de fleste patienter med sle har antistoffer i blodet, som er rettet mod dobbeltstrenget dna (deoxyribonukleinsyre).
most patients with sle have antibodies in their blood that are directed against double-stranded dna (deoxyribonucleic acid).
azacitidin indbygges i cellernes genetiske materiale (ribonukleinsyre (rna) og deoxyribonukleinsyre (dna).
azacitidine becomes incorporated into the genetic material of cells (ribonucleic acid (rna) and deoxyribonucleic acid (dna)).
ligesom for andre midler i gruppen af deoxyribonukleinsyre- (dna-) beskadigende midler kan behandling med pixuvri være forbundet med nedsat fertilitet.
as with other agents in the general class of deoxyribonucleic acid (dna) damaging agents, pixuvri may be associated with fertility impairment.
i lyset af påvirkningen af cellernes deoxyribonukleinsyre (dna) er 6–mercaptopurin potentielt kræftfremkaldende, og denne behandlings teoretiske risiko for carcinogenese bør tages i betragtning.
in view of its action on cellular deoxyribonucleic acid (dna) 6–mercaptopurine is potentially carcinogenic and consideration should be given to the theoretical risk of carcinogenesis with this treatment.
arsentrioxid fremkalder morfologiske ændringer og deoxyribonukleinsyre (dna)-fragmenteringsegenskaber, som er karakteristisk for apoptose ved nb4-humane promyelocyt leukæmi-celler in vitro.
arsenic trioxide causes morphological changes and deoxyribonucleic acid (dna) fragmentation characteristic of apoptosis in nb4 human promyelocytic leukaemia cells in vitro.
akkumulering af ara-gtp i leukæmiske stamceller giver mulighed for selektiv indarbejdning af ara- gtp i deoxyribonukleinsyre (dna), som fører til hæmning af dna-syntesen.
accumulation of ara-gtp in leukaemic blasts allows for preferential incorporation of ara-gtp into deoxyribonucleic acid (dna) leading to inhibition of dna synthesis.