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glucuronidering niet significant remmen of induceren
be used with other medicinal (once a day) products that do not significantly inhibit or induce lamotrigine
in vitro remt sorafenib glucuronidering via ugt1a1 en ugt1a9.
in vitro, sorafenib inhibited glucuronidation via ugt1a1 and ugt1a9.
glucuronidering is de belangrijkste metabolische route voor deferasirox, met daaropvolgend biliaire uitscheiding.
glucuronidation is the main metabolic pathway for deferasirox, with subsequent biliary excretion.
dolutegravir wordt primair gemetaboliseerd door middel van glucuronidering via ugt1a1 met een kleine cyp3a-component.
dolutegravir is primarily metabolized through glucuronidation via ugt1a1 with a minor cyp3a component.
abacavir, zidovudine: concentraties kunnen afnemen vanwege toegenomen glucuronidering door lopinavir/ritonavir.
abacavir, zidovudine: concentrations may be reduced due to increased glucuronidation by lopinavir/ritonavir.
stoppen van geneesmiddelen die niet significant lamotrigine-glucuronidering remmen of induceren (zie rubriek 4.5):
withdrawal of medicinal products that do not significantly inhibit or induce lamotrigine glucuronidation (see section 4.5):
regorafenib wordt voornamelijk in de lever gemetaboliseerd, door cyp3a4-gemedieerde oxidatief metabolisme en door ugt1a9-gemedieerde glucuronidering.
regorafenib is metabolized primarily in the liver by oxidative metabolism mediated by cyp3a4, as well as by glucuronidation mediated by ugt1a9.