Vous avez cherché: glucose 6 phosphate dehydrogenase (Anglais - Slovène)

hexokinase/glucose 6-phosphate dehydrogenase:
heksokinaza/glukoza 6-fosfat dehidrogenaza:

patients with glucose-6-phosphate dehydrogenase deficiency
bolniki s pomanjkanjem glukoza-6-fosfat-dehidrogenaze

glucose-6-phosphate dehydrogensae deficiency
pomanjkanje glukoza-6-fosfat-dehidrogenaze

- if you or any member of your family have glucose-6-phosphate dehydrogenase deficiency (a
- Če imate vi ali katerikoli član vaše družine pomanjkanje glukoza- 6- fosfat dehidrogenaze (redko

- patients with a family history of, or actual glucose-6-phosphate dehydrogenase deficiency are
- ker so bolniki, ki imajo pomanjkanje glukoza- 6- fosfat dehidrogenaze ali pa je ta motnja

patients with glucose-6-phosphate dehydrogenase deficiency (g6pd) due to the risk of haemolytic anaemia
bolniki s pomanjkanjem glukoza-6-fosfat-dehidrogenaze (g6pd) zaradi tveganja hemolitične anemije.

it must not be used in patients with the following conditions: glucose-6-phosphate dehydrogenase deficiency (g6pd),
zdravila ne smejo uporabljati bolniki z naslednjimi vrstami obolenj: pomanjkanjem glukoza-6-fosfat dehidrogenaze (g6pd),

failure to respond to methylthioninium chloride suggests cytochrome b5 reductase deficiency, glucose-6- phosphate dehydrogenase deficiency or sulfhaemoglobinemia.
neodzivanje na metiltioninijev klorid kaže na pomanjkanje citokrom-b5-reduktaze, pomanjkanje glukoza-6-fosfat-dehidrogenaze ali sulfhemoglobinemijo.

glucose-6-phosphate dehydrogenase (g6pd) deficiency and other cellular metabolic disorders known to cause haemolysis and methemoglobinemia.
pomanjkanje glukoze-6-fosfat dehidrogenaze (g6pd) ali druge celularne presnovne motnje, za katere je znano, da povzročajo hemolizo in methemoglobinemijo.

glucose-6-phosphate dehydrogenase defect (hereditary disease of the red blood cells based on a defect in an enzyme).
okvara glukoza- 6- fosfat dehidrogenaze (dedna bolezen eritrocitov, ki temelji na okvari encima).

glucose-6-phosphate dehydrogenase deficiency haemolytic reactions have been reported with ciprofloxacin in patients with glucose-6-phosphate dehydrogenase deficiency.
pomanjkanje glukoza– 6– fosfat dehidrogenaze pri bolnikih s pomanjkanjem glukoza– 6– fosfat dehidrogenaze, ki so prejemali ciprofloksacin, so poročali o hemolitičnih reakcijah.

patients with latent or actual defects in glucose-6-phosphate dehydrogenase activity may be prone to haemolytic reactions when treated with quinolone antibacterial agents.
bolniki z latentnimi ali izraženimi motnjami aktivnosti glukoza-6-fosfat-dehidrogenaze so med zdravljenjem s kinolonskimi protibakterijskimi zdravili lahko nagnjeni k hemolitičnim reakcijam.

g6p + nadp+ gluconate 6-phosphate + nadph + h+
g6p + nadp+ glukonat-6-fosfat + nadph + h+

they can produce adverse biological effects including hypokalaemia, haemolysis (glucose-6-phosphate dehydrogenase (g6pd) defect) and hypersensitivity due to sulphonamide properties.
lahko povzročajo neželene biološke učinke, med drugim hipokaliemijo, hemolizo (nepravilnost glukoza- 6- fosfat dehidrogenaze (g6pd)) in preobčutljivost zaradi sulfonamidnih lastnosti.

suspension hk/g6p-dh [hexokinase (ec 2.7.1.1) and glucose-6-phosphate-dehydrogenase (ec 1.1.1.49)]:
suspenzija hk/g6p-dh (heksokinaza (ec 2.7.1.1) in glukoza-6-fosfat-dehidrogenaza (ec 1.1.1.49):

at the end of this reaction, the fructose 6-phosphate is converted into glucose 6-phosphate by the action of phosphoglucose isomerase (pgi):
na koncu te reakcije fruktoza 6-fosfat preide v glukozo 6-fosfat z delovanjem fosfoglukoze izomeraze (pgi):

your doctor will check your renal function carefully. • glucose-6-phosphate dehydrogenase defect (hereditary disease of the red blood cells based on a defect in an enzyme).
zdravnik bo pozorno pregledal vaše ledvice; • okvara glukoza- 6- fosfat- dehidrogenaze (dedna bolezen eritrocitov, katere vzrok je okvara encima).

• tell your doctor if you or a member of your family is known to have a deficiency in glucose-6- phosphate dehydrogenase (g6pd), since you may experience a risk of anemia with ciprofloxacin.
intravenska uporaba pred uporabo preberite priloženo navodilo.

idursulfase is taken up by selective receptor-mediated mechanisms involving binding to mannose 6- phosphate receptors.
prevzem idursulfaze poteka prek mehanizmov, ki jih uravnavajo selektivni receptorji, pri katerih se zdravilo veže na receptorje za manozo-6-fosfat.

the glucose 6-phosphate is first oxidized to gluconate 6-phosphate by nicotinamide adenine dinucleotide phosphate (nadp) in the presence of the enzyme glucose 6-phosphate dehydrogenase (g6pdh).
glukoza-6-fosfat najprej oksidira v glukonat-6-fosfat z nikotinamidadenindinukleotidfosfatom (nadp) v navzočnosti encima glukoza 6-fosfat dehidrogenaza (g6pdh).