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Hindi

department of state for visa processing

English

 

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Hindi (indiano)

Inglese

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Hindi (indiano)

mai out of state hu 4december tak

Inglese

mai out of state hu 4 december tak

Ultimo aggiornamento 2023-11-30
Frequenza di utilizzo: 1
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Hindi (indiano)

we have to invest ok so we can have profile let me use it for visa

Inglese

do you want me to come over there

Ultimo aggiornamento 2023-11-20
Frequenza di utilizzo: 1
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Hindi (indiano)

who envies none that chance doth raise .nor vice who never understood .how deepest wounds are given by prasie.nor rules of state but rule of good

Inglese

who hath his life from rumour freed . whose conscience is his strong retreat . whose state can neither flatterers feed ,. nor ruin make oppressor great

Ultimo aggiornamento 2023-05-17
Frequenza di utilizzo: 1
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Hindi (indiano)

hello, i am dirya from the hr department of influence marketing hub. we are hiring freelancers who can "work from home" so sorry to bother you i will like to know if you are free we can talk more about the job.

Inglese

Ultimo aggiornamento 2023-07-22
Frequenza di utilizzo: 1
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Attenzione: contiene formattazione HTML nascosta

Hindi (indiano)

मध्य पूर्व अध्ययन के क्षेत्र में अमेरिका समर्थक और इजरायल समर्थक कुछ आवाजों में से एक आवाज होने के चलते इस क्षेत्र के अन्य लोगों से मुझे प्रायः अलग थलग कर दिया जाता है । इसी कारण मुझे अपनी वेबसाइट पर 5 , 000 शब्दों का एक दस्तावेज प्रकाशित करना पडा जिसका शीर्षक है ” department of corrections

Inglese

muslims have gone through a trauma during the last two hundred years - the tribulation of god ' s people who unaccountably found themselves at the bottom of the heap . the strains have been enormous and the results agonizing ; muslim countries have the most terrorists and the fewest democracies in the world . only turkey is fully democratic , and even there the system is frail . everywhere else , the head of government got to power through force his own or someone else ' s . the result is endemic instability plus a great deal of aggression .

Ultimo aggiornamento 2020-05-24
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Hindi (indiano)

duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long prote “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.

Inglese

duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long proteins can have different localization, stability and function. “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.

Ultimo aggiornamento 2021-03-15
Frequenza di utilizzo: 1
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Riferimento: Anonimo

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