検索ワード: dna polymerase (英語 - スロベニア語)

nucleoside analogue dna polymerase inhibitors
nukleozidni analogi - zaviralci dnk-polimeraze

10 x buffer (furnished with the taq dna polymerase)
10 x pufer (priložen s taq dna polimerazo)

mgcl2 (furnished with the dna polymerase) (25 mm)
mgcl2 (priložen z dna polimerazo) (25 mm)

the entecavir-tp ki for hbv dna polymerase is 0.0012 μm.
ki entekavir trifosfata za hbv dna polimerazo je 0,0012 μm.

the entecavir-tp ki for hbv dna polymerase is 0.0012 μ m.
ki entekavir trifosfata za hbv dna polimerazo je 0, 0012 μm.

dna polymerase) that are essential for the viruses to reproduce themselves.
tenofovir je nukleotidni zaviralec reverzne transkriptaze, ki je splošno znan pod imenom nukleozidni zaviralci reverzne transkriptaze (nrti, nucleoside reverse transcriptase inhibitors), in deluje tako, da vpliva na normalno delovanje encimov (reverzne transkriptaze pri virusu hiv in dna- polimeraze pri virusu hbv), ki sta bistvena za pomnoževanje virusa.

telbivudine is a synthetic thymidine nucleoside analogue with activity against hbv dna polymerase.
telbivudin je sintetični timidinski nukleozidni analog, ki deluje na dna polimerazo virusa hepatitisa

rilpivirine does not inhibit the human cellular dna polymerases α, β and mitochondrial dna polymerase γ.
rilpivirin ne zavira humanih celičnih dna polimeraz α, β in mitohondrijske dna polimeraze γ.

emtricitabine is a weak inhibitor of mammalian dna polymerase α, β and ε and mitochondrial dna polymerase γ.
emtricitabin je šibek zaviralec sesalske dna-polimeraze α, β in ε ter mitohondrijske dna-polimeraze γ.

no hbv dna polymerase mutations associated with resistance to adefovir were identified when patients were genotyped at baseline and at week 48.
pri genotipiziranju bolnikov na začetku in v 48. tednu ni bilo identificiranih nobenih hbv dna-polimeraznih mutacij povezanih z rezistenco proti adefovirju.

telbivudine interferes with the action of a viral enzyme called dna polymerase, which is involved in the formation of viral dna.
telbivudin zavira delovanje virusnega encima, imenovanega dnk-polimeraza, ki je udeležen pri tvorbi virusne dnk.

intracellularly, decitabine is activated through sequential phosphorylation via phosphokinase activities to the corresponding triphosphate, which is then incorporated by the dna polymerase.
v celici fosfokinaze s sekvenčno fosforilacijo aktivirajo decitabin v ustrezen trifosfat, tega pa nato dna polimeraze vgradijo v dna.

cidofovir-selected mutants had a mutation in the dna polymerase gene and were cross-resistant to ganciclovir, but susceptible to foscarnet.
s cidofovirom selekcionirani mutanti so imeli mutacijo v genu za dna polimerazo in so bili navzkrižno odporni proti gancikloviru, a občutljivi za foskarnet.

the hepatitis b virus, for example, carries its genetic material in the form of dna, and employs a rna-dependent dna polymerase to replicate.
cev je leta 1857 izumil nemški fizik in steklar heinrich geissler.

it is also only a weak inhibitor of mammalian dna polymerases alpha and beta.
prav tako je le šibek zaviralec dna polimeraz alfa in beta pri sesalcih.

abacavir and lamivudine triphosphates show significantly less affinity for host cell dna polymerases.
abakavir in lamivudin trifosfat imata občutno manjšo afiniteto do dna polimeraz gostiteljske celice.

nevirapine binds directly to reverse transcriptase and blocks the rna-dependent and dna-dependent dna polymerase activities by causing a disruption of the enzyme's catalytic site.
veže se direktno na reverzno transkriptazo in blokira od rna in dna odvisne učinke dna- polimeraze, tako da razgradi katalitičo mesto encima.

abacavir, lamivudine and zidovudine triphosphates show significantly less affinity for host cell dna polymerases.
abakavir, lamivudin in zidovudin trifosfati imajo občutno manjšo afiniteto do dnk polimeraz gostiteljske celice.

both emtricitabine triphosphate and tenofovir diphosphate are weak inhibitors of mammalian dna polymerases and there was no evidence of toxicity to mitochondria in vitro and in vivo.
emtricitabin trifosfat in tenofovir difosfat sta šibka zaviralca sesalske dna-polimeraze in toksičnost za mitohondrije pri in vitro ali in vivo pogojih ni bila dokazana.

in the body, this active analogue takes the place of guanine and interferes with the enzymes involved in making new dna, dna polymerases.
dna polimeraze.