전문 번역가, 번역 회사, 웹 페이지 및 자유롭게 사용할 수 있는 번역 저장소 등을 활용합니다.
in dogs, the decreased cardiac contractility and hypotension responded to acute intervention with positive inotropic or pressor agents.
fi klieb it-tnaqqis fil-kapaċità kardijaka ta’ kontrazzjoni u ipotensjoni rrispondew għall-intervent akut b’sustanzi inotropiċi pożittivi jew pressor.
the circulatory changes included decreased blood pressure and, in some studies, increased heart rate and decreased contractility.
il-bidliet ċirkolatorji kienu jinkludu pressjoni mnaqqsa tad-demm u f’xi studji ġie osservat ukoll żjieda fir-rata tat-taħbit tal-qalb u nuqqas ta’ kontrazzjoni.
replagal was also associated with improved myocardial contractility, a decrease in mean qrs duration and a concomitant decrease in septal thickness on echocardiography.
replagal kien ukoll assoċjat ma’ titjib fil-kontratilità mijokardjali, tnaqqis fid-dewmien qrs medju u tnaqqis konkomitanti tal-ħxuna septali fuq l-ekokardjografija.
ivabradine does not influence intracardiac conduction, contractility (no negative inotropic effect) or ventricular repolarisation:
ivabradine m’ għandux effett fuq il- ħin biex igħaddi l- istimulu intrakardijaku, fuq il- kontrazzjoni (l- ebda effett negattiv inotropiku) u l- anqas fuq ir- ripolarizzazzjoni ventrikulari:
the pimobendan-induced increase in contractility of the heart is attenuated in the presence of the calcium antagonist verapamil and the ß-antagonist propranolol.
iż-żieda fil-kontrattilità tal-qalb, ikkawżata minn pimobendan, titnaqqas fil-preżenza tal- antagonist tal-kalċju, verapamil, u l-antagonist-ß, propranolol.
blockade of these α1a-adrenoreceptors causes smooth muscle in these tissues to relax, thus decreasing bladder outlet resistance, without affecting detrusor smooth muscle contractility.
l-imblokk ta’ dawn l-adrenoriċetturi-α1a jġiegħel il-muskolu lixx f’dawn it-tessuti li jirrilassa, u b’hekk inaqqas ir-reżistenza tal-iżbokk tal-bużżieqa tal-awrina, mingħajr ma jaffettwa l-kontratilità tal-muskolu lixx detrusor.
dronedarone decreases arterial blood pressure and myocardial contractility (dp/dt max) with no change in left ventricular ejection fraction and reduces myocardial oxygen consumption.
dronedarone inaqqas il-pressjoni arterjali tad-demm u l- kontrattilità mijokardika (dp/dt max) bla ebda tibdil fil-volum li jiġi mneħħi mill-ventriklu tax-xellug u jnaqqas il-konsum tal-ossiġenu min-naħa tal-mijokardju.
in particular, sildenafil has greater than 4,000-fold selectivity for pde5 over pde3, the camp-specific phosphodiesterase isoform involved in the control of cardiac contractility.
b’mod partikulari, sildenafil għandu selettività ta’ aktar minn 4,000-darba għal pde5 fuq pde3, is-camp –specific phosphodiesterase isoform involut fil- kontroll tal-kontrattilità tal-qalb.
each single agent produced effects on left ventricular contractility and pr prolongation at concentrations at least 35-fold higher than the free atazanavir and cobicistat concentrations at the recommended human dose (rhd) cmax.
kull sustanza waħdiena pproduċiet effetti fuq il-kontrattilità ventrikulari tax-xellug u titwil ta’ pr f’konċentrazzjonijiet tal-anqas 35 darba ogħla mill- konċentrazzjonijiet ħielsa ta’ atazanavir u cobicistat fis-cmax tad-doża rakkomandata fil-bniedem (rhd).
if during pregnancy the woman is already breast-feeding an earlier child, then breast-feeding should be discontinued during treatment with atosiban, since the release of oxytocin during breast-feeding may augment uterine contractility, and may counteract the effect of tocolytic therapy.
jekk waqt it-tqala l-omm tkun diġà qed tredda’ tarbija oħra li kellha qabel, it-treddigħ ghandu jitwaqqaf waqt it-trattament bi atosiban, dan għaliex ir-rilaxx ta’ oxytocin waqt it-treddigħ jista’ jżzid il-kontrazzjonijiet tal-utru, u jista’ jaħdem kontra l-effett tat-terapija tokolitika.