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hypertenzia hypotenzia vazokonstrikcia bledosť
hypertension hypotension vasoconstriction pallor
Son Güncelleme: 2017-04-26
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v spojivkovom obehu a iných očných cievach vzniká vazokonstrikcia v miere vystavenia lieku.
vasoconstriction occurs in the conjunctival circulation and in other ocular vessels to the extent that they are exposed to drug.
Son Güncelleme: 2017-04-26
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benazepril, tým, že zabraňuje vzniku angiotenzínu-ii, bráni nežiaducim účinkom ako je vazokonstrikcia astimulácia uvoľňovania aldosterónu.
benazepril, by preventing the formation of angiotensin-ii, inhibits the detrimental effects of vasoconstriction and stimulation of aldosterone release.
Son Güncelleme: 2017-04-26
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benazepril, tým, že zabraňuje vzniku angiotenzínu-ii, bráni nežiaducim účinkom ako je vazokonstrikcia a stimulácia uvoľňovania aldosterónu.
benazepril, by preventing the formation of angiotensin-ii, inhibits the detrimental effects of vasoconstriction and stimulation of aldosterone release.
Son Güncelleme: 2017-04-26
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po náhlom vysadení inhalačnej liečby oxidom dusnatým boli opísané rýchle rebound reakcie, napríklad zvýšená pulmonálna vazokonstrikcia a hypoxia; precipitačný kardiovaskulárny kolaps.
rapid rebound reactions such as intensified pulmonary vasoconstriction and hypoxia after sudden withdrawal of inhaled nitric oxide therapy has been described, precipitating cardiovascular collapse.
Son Güncelleme: 2017-04-26
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angiotenzín ii je hlavná presorická látka raas, medzi ktorého účinky patria vazokonstrikcia, stimulácia syntézy a uvoľňovanie aldosterónu, stimuláciu srdca a renálna reabsorpcia sodíka.
angiotensin ii is the principal pressor agent of the raas, with effects that include vasoconstriction, stimulation of synthesis and release of aldosterone, cardiac stimulation, and renal reabsorption of sodium.
Son Güncelleme: 2017-04-26
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stimulácia receptora at1 je zodpovedná za patologické účinky angiotenzínu ii v obličkách a iných orgánoch súvisiacich s angiotenzínom ii, ako je vazokonstrikcia, zadržiavanie sodíka a vody, zvýšená syntéza aldosterónu a zmeny v orgánoch.
stimulation of the at1 receptor is responsible for pathologic effects of angiotensin ii in the kidney and other organs associated with angiotensin ii such as vasoconstriction, retention of sodium and water, increased aldosterone synthesis and organ remodeling.
Son Güncelleme: 2017-04-26
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angiotenzín ii je hlavná presorická látka renín- angiotenzínového systému, medzi ktorého účinky patria vazokonstrikcia, stimulácia syntézy a uvoľňovanie aldosterónu, stimuláciu srdca a renálna reabsorpcia sodíka.
angiotensin ii is the principal pressor agent of the renin-angiotensin system, with effects that include vasoconstriction, stimulation of synthesis and release of aldosterone, cardiac stimulation, and renal reabsorption of sodium.
Son Güncelleme: 2017-04-26
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endotelín (et)-1 a jeho receptory (eta a etb) sprostredkúvajú viacero účinkov, ako je vazokonstrikcia, fibróza, proliferácia, hypertrofia a zápal.
endothelin (et)-1 and its receptors (eta and etb) mediate a variety of effects such as vasoconstriction, fibrosis, proliferation, hypertrophy, and inflammation.
Son Güncelleme: 2017-04-26
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