Şunu aradınız:: mitochondrial (İngilizce - Maltaca)

mitochondrial toxicity
tossiċità mitokondrijali

mitochondrial toxicity.
żewġ mediċini f’ daqqa jkun limitat.

mitochondrial dysfunction:
funzjoni mitokondrijali ħażina:

mitochondrial toxicity and lactic acidosis
tossiċità mitokondrijali u aċidożi lattika

mitochondrial toxicity and lactic acidosis:
infezzjoni fl- istess ħin b’ hcv/ hiv tossiċità mitokondrijali u aċidożi lattika:

tedizolid inhibits mitochondrial protein synthesis.
tedizolid jinibixxi s-sinteżi tal-proteini mitokondrijali.

mitochondrial dysfunction following exposure in utero
disfunzjoni mitokondrija wara esponiment in utero

mitochondrial toxicity (see section 4.5).
mitokondrijali (ara sezzjoni 4. 5).

description of selected adverse reactions mitochondrial toxicity
deskrizzjoni ta’ reazzjonijiet avversi magħżula tossiċità mitokondrijali

rna polymerases nor an inhibitor of mitochondrial rna polymerase.
rna polymerases, u lanqas mhu inibitur ta’ rna polymerase mitokondrijali.

the effect of bosentan on mitochondrial function is unknown.
l-effett ta’ bosentan fuq il-funzjoni tal-mitokondrija mhuwiex magħruf.

hcv/hiv co-infection mitochondrial toxicity and lactic acidosis:
infezzjoni fl- istess ħin b’ hcv/ hiv tossiċità mitokondrijali u aċidożi lattika:

the mitochondrial membrane is more readily permeable for carglumic acid than for n- acetylglutamate
ir-rita mitokondrijali hi aktar permeabbli għal carglumic acid milli għal n-acetylglutamate.

rilpivirine does not inhibit the human cellular dna polymerases α, β and mitochondrial dna polymerase γ.
rilpivirine ma jimpedixxix il-polymerases α u β tad-dna ċellulari u l-polymerase γ tad-dna mitkondrijali tal-bniedem.

emtricitabine is a weak inhibitor of mammalian dna polymerase α, β and ε and mitochondrial dna polymerase γ.
emtricitabine hija impeditur dgħajjef tal-polimerat tad-dna mammifera α, β u ε u polimerat mitokondrijali tad-dna γ.

nucleoside and nucleotide analogues have been demonstrated in vitro and in vivo to cause a variable degree of mitochondrial damage.
analogi nucleoside u nucleotide intwerew in vitro u in vivo li jikkawżaw grad varjabbli ta’ ħsara mitokondrijali.

the source and method for obtaining the nucleic acid of interest (cdna, chromosomal mitochondrial, etc.);
is-sors u l-metodu għall-ksib ta' l-aċidu nuklejiku rilevanti (cdna, kromożomiku, mitokondriku, eċċ.);

in assays relating to potential substance effects on mitochondrial structure and dna content and function, lamivudine lacked appreciable toxic effects.
fl- analiżi ta 'l- effetti possibbli tas- sustanza fuq struttura mitochondrial u l- kontenut u l- funzjoni ta' dna, lamivudine ma nstabx li kellu xi effetti serji ta 'tossiċita'.

mitochondrial loss/damage was also observed; however the role and significance of this finding in the overall pathology are unknown.
telf/ħsara fil-mitokondrija kien osservat ukoll, madankollu r-rwol u s-sinifikat ta’ din is-sejba fil-patoloġija globali mhumiex magħrufa.

a second mitochondrial oxidoreductase enzyme, a transhydrogenase, also catalyses the conversion to succinic semialdehyde in the presence of -ketoglutarate.
enżim tal-mitokondrion oxidoreductase ieħor, li hu transhydrogenase, jikkatalizza ukoll il-konverżjoni f’ succinic semialdehyde fil-preżenza ta’ -ketoglutarate.