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rilutek negalima vartoti pacientams, sergantiems kitomis motorini neuron ligomis.
rilutek should not be used in patients with any other form of motor neurone disease.

datscan skirtas funkcini dopaminergini neuron terminal netekimui dryzuotame kne diagnozuoti:
datscan is indicated for detecting loss of functional dopaminergic neuron terminals in the striatum:

nkk blokatoriai reguliuoja neurotransmiteri issiskyrim specifinse neuron populiacijose, atsakingose uz skausmo perdavim
nccs regulate neurotransmitter release in specific neuronal populations responsible for the spinal processing of pain.

gydym rilutek gali pradti tik gydytojas, turintis motorini neuron lig gydymo patirties.
treatment with rilutek should only be started by a specialist doctor with experience in the management of motor neurone diseases.

nuolatinio n tipo kalcio kanal blokavimo ilgalaiki pasekmi neuron veiklai bandomiesiems gyvnams nenustatyta.
the long-term consequences to neuronal function of continuous n-type calcium-channel block have not been demonstrated in experimental animals.

nkk blokatoriai reguliuoja neurotransmiteri issiskyrim specifinse neuron populiacijose, atsakingose uz skausmo perdavim nugaros smegenyse.
nccs regulate neurotransmitter release in specific neuronal populations responsible for the spinal processing of pain.

deksmedetomidinas yra stiprus ir selektyvus 2-adrenoceptori agonistas, slopinantis noradrenalino issiskyrim is noradrenergini neuron.
dexmedetomidine is a potent and selective 2-adrenoceptor agonist that inhibits the release of noradrenaline from noradrenergic neurons.

rilutek saugumas ir veiksmingumas tirtas tik lateraline amiotrofine skleroze sergantiems pacientams, todl kitoms motorini neuron ligoms gydyti sis vaistas netinka.
safety and efficacy of rilutek has only been studied in als.therefore, rilutek should not be used in patients with any other form of motor neurone disease.

rivastigminas yra karbamato tipo acetilcholinesterazs ir butirilcholinesterazs inhibitorius, pagerinantis cholinerginio impulso perdavim neuronais, neleisdamas skaidyti sveik neuron isskirt acetilcholin.
rivastigmine is an acetyl-and butyrylcholinesterase inhibitor of the carbamate type, thought to facilitate cholinergic neurotransmission by slowing the degradation of acetylcholine released by functionally intact cholinergic neurones.

rivastigminas yra karbamato tipo acetilcholinesterazs ir butirilcholinesterazs inhibitorius, pagerinantis cholinerginio impulso perdavim neuronais, neleisdamas skaidyti sveik cholinergini neuron isskirt acetilcholin.
rivastigmine is an acetyl-and butyrylcholinesterase inhibitor of the carbamate type, thought to facilitate cholinergic neurotransmission by slowing the degradation of acetylcholine released by functionally intact cholinergic neurones.

paroksetinas yra fenilpiperidino darinys ir selektyvaus poveikio priessinapsinio 5-hidroksitriptamino (5-ht) patekimo neuron inhibitorius.
paroxetine is a phenylpiperidine derivative and is a selective inhibitor of the presynaptic 5-hydroxytryptamine (5-ht) reuptake.

manoma, kad sergant motorini neuron liga nerv lsteles naikina per didelis neurotransmiterio gliutamato (chemins medziagos, perduodancios informacij) kiekis.
it is thought that the destruction of nerve cells in motor neurone disease may be caused by too much glutamate, a neurotransmitter (chemical messenger) .

zonisamidas taip pat turi moduliacin poveik gama aminosviesto rgsties (gasr, angl. gama -aminobutyric acid, gaba) slygojamam neuron slopinimui.
zonisamide also has a modulatory effect on gaba-mediated neuronal inhibition.

lateralins amiotrofins sklerozs patogenez pilnutinai neistirta, taciau manoma, kad sia serganci pacient neuron zuvimui turi reiksms glutamatas (pagrindinis stimuliuojantis neuromediatorius cns) .
although the pathogenesis of als is not completely elucidated, it is suggested that glutamate (the primary excitatory neurotransmitter in the central nervous system) plays a role for cell death in the disease.

elektrofiziologiniai tyrimai parod, kad olanzapinas selektyviai mazina mezolimbini (a10) dopaminergini neuron iskrov ir beveik neveikia motorines funkcijas reguliuojancios dryzuotojo kno (a9) sistemos.
electrophysiological studies demonstrated that olanzapine selectively reduced the firing of mesolimbic (a10) dopaminergic neurons, while having little effect on the striatal (a9) pathways involved in motor function.