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učinki hialuronidaze so reverzibilni in permeabilnost subkutanega tkiva se povrne v 24 do 48 urah.
the effects of the hyaluronidase are reversible and permeability of the subcutaneous tissue is restored within 24 to 48 hours.
rekombinantna humana hialuronidaza je topna rekombinantna oblika humane hialuronidaze, ki poveča permeabilnost subkutanega tkiva, tako da začasno depolimerizira hialuronan.
recombinant human hyaluronidase is a soluble recombinant form of human hyaluronidase that increases the permeability of the subcutaneous tissue by temporarily depolymerizing hyaluronan.
in vivo je uporaba vandetaniba zmanjšala tumorsko celično inducirano angiogenezo, permeabilnost tumorskih žil, gostoto tumorskega mikrožilja ter zavrla rast tumorjev v celi vrsti modelov ksenotransplantatov človeških tumorjev pri atimičnih miših.
in vivo vandetanib administration reduced tumour cell-induced angiogenesis, tumour vessel permeability, tumour microvessel density, and inhibited tumour growth of a range of human xenograft tumour models in athymic mice.
glede na biofarmacevtski klasifikacijski sistem (biopharmaceutics classification system) spada vemurafenib v razred iv (nizka topnost in permeabilnost).
vemurafenib is a class iv substance (low solubility and permeability), using the criteria described in the biopharmaceutics classification system.
dejavniki, ki vplivajo na vzajemno indukcijo: število ovojev tuljave, velikost tuljave, permeabilnost tuljave, položaj tuljav v odnosu druga do druge;
factors affecting mutual inductance: number of turns in coil, physical size of coil, permeability of coil, position of coils with respect to each other;
vezava vegf-a na njegove receptorje povzroča proliferacijo endotelijskih celic in neovaskularizacijo, pa tudi povečano žilno permeabilnost, kar vse prispeva k razvoju neovaskularne oblike starostne degeneracije makule, degenerativne kratokovidnosti ali k okvari vida zaradi diabetičnega makularnega edema ali zaradi makularnega edema pri zapori mrežnične vene.
binding of vegf-a to its receptors leads to endothelial cell proliferation and neovascularisation, as well as vascular leakage, all of which are thought to contribute to the progression of the neovascular form of age-related macular degeneration, pathologic myopia or to visual impairment caused by either diabetic macular oedema or macular oedema secondary to rvo.