Sie suchten nach: he is first who call me bua (Englisch - Hindi)

call me bua
वह मुझे बुआ कहते थे

she call me bua
मेरी राजकुमारी मुझे बुआ बुलाती है

i miss her who call me
अगर आपको मेरी याद आती है तो आप मुझे कॉल कर सकते हैं

i m waiting for that day when he will call me bua
उस दिन की प्रतीक्षा कर रहा हूं जब मैं तुम्हें याद नहीं करूंगा

he is in meeting but customer don't call me back
वह बैठक में है, लेकिन वापस नहीं बुलाता

i m waiting for that day when you call me bua
उस दिन की प्रतीक्षा कर रहा हूं जब मैं तुम्हें याद नहीं करूंगा

he is first player to make double century in one day international cricket .
एकदिवसीय अंतरराष्ट्रीय क्रिकेट के इतिहास में दोहरा शतक जड़ने वाले पहले खिलाड़ी बने

there are people who call me and think of me .
कई लोग मुझे पुकारते हैं और मेरे बारे में सोचते हैं ।

call me when he is not there
आप लोगों ने की थी चर्चा

don't call me brother i have 2 brothers and he is brother for me
don't call me brother i have 2 brothers and he is brother for me.

at first he is under the patronage of a rao bahadur who calls himself a social reformer and is a supporter of the british rule .
पहले वह एक रायबहादुर की संरक्षण में रहता है जो अपने को समाज - सुधारक कहते हैं और ब्रिटिश शासन के हिमायती हैं ।

so send my brother aaron with me as helper for he is more fluent than i with words , that he should affirm me , for i fear that they would call me a liar . "
और मेरा भाई हारुन वह मुझसे फ़सीह है तो तू उसे मेरे साथ मेरा मददगार बनाकर भेज कि वह मेरी तसदीक करे क्योंकि यक़ीनन मै इस बात से डरता हूँ कि मुझे वह लोग झुठला देंगे

very sorry to hear that your father is no more.but what to do.god is very selfish.he call them who he is love.faith on god.everything will be alright.if you are in any problem call me once..iam always there for you
यह सुनकर बहुत दुख हुआ कि तुम्हारे पिता नहीं रहे।पर क्या करें।भगवान बहुत स्वार्थी हैं।वह उन्हें बुलाते हैं जिन्हें वह प्यार करते हैं।ईश्वर पर विश्वास।सब ठीक हो जाएगा।यदि आप किसी भी समस्या में हैं तो मुझे एक बार कॉल करें।मैं आपके लिए सदैव

duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long proteins can have different localization, stability and function. “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.
duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long prote “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.