Sie suchten nach: polymerases (Maltesisch - Englisch)

dna polymerases α, β, γ jew δ tal- bniedem).
hiv-2 reverse transcriptase and eukaryotic dna polymerases (such as human dna polymerases α, β, γ, or δ) are not inhibited by nevirapine.

rna polymerases, u lanqas mhu inibitur ta’ rna polymerase mitokondrijali.
rna polymerases nor an inhibitor of mitochondrial rna polymerase.

hu wkoll inibitur verament dgħajjef tad-dna polymerases mammiferi alfa u beta.
it is also only a weak inhibitor of mammalian dna polymerases alpha and beta.

rilpivirine ma jinibixxix il- polymerases α, β u γ tad-dna taċ-ċelluli umani.
rilpivirine does not inhibit the human cellular dna polymerases α, β and γ.

abacavir u lamivudine triphosphates juru affinita’ sostanzjalment inqas għal polymerases tad-dna taċ-ċellula reċipjenta.
abacavir and lamivudine triphosphates show significantly less affinity for host cell dna polymerases.

rilpivirine ma jinibixxix il-polymerases ta’ dna ċellulari bniedem α, β u polymerase ta’ dna mitokondrijali γ.
rilpivirine does not inhibit the human cellular dna polymerases α, β and mitochondrial dna polymerase γ.

abacavir, lamivudine u zidovudine triphosphates juru ferm inqas affinita 'ma ’ polymerases tad- dna taċ- ċelluli li jkunu qegħdin fihom.
abacavir, lamivudine and zidovudine triphosphates show significantly less affinity for host cell dna polymerases.

kemm emtricitabine triphosphate u tenofovir diphosphate huma inibituri dgħajfa ta’ polymerases tad- dna tal-mammali u ma kien hemm ebda evidenza ta’ tossiċità għal mitokondrija in vitro u in vivo.
both emtricitabine triphosphate and tenofovir diphosphate are weak inhibitors of mammalian dna polymerases and there was no evidence of toxicity to mitochondria in vitro and in vivo.