Results for neovaskularisering translation from Danish to English

myopisk koroidal neovaskularisering
myopic choroidal neovascularisation

visudyne anvendes til behandling af subfoveal koroidal neovaskularisering.
visudyne is used for the treatment of patients who have subfoveal choroidal neovascularisation.

der findes to former for choroidal neovaskularisering: klassisk og okkult.
there are two types of choroidal neovascularisation: classic and occult.

vegf165 er den vegf isoform, der fortrinsvis indgår i patologisk okulær neovaskularisering.
vegf165 is the vegf isoform preferentially involved in pathological ocular neovascularisation.

plgf er også forbundet med patologisk neovaskularisering og rekruttering af inflammatoriske celler til tumorer.
plgf is also linked to pathological neovascularisation and recruitment of inflammatory cells into tumours.

i de to undersøgelser med klassisk subfoveal koroidal neovaskularisering var visudyne mere effektivt end placebo.
in the two studies of classic subfoveal choroidal neovascularisation, visudyne was more effective than placebo.

til patienter med koroidal neovaskularisering forårsaget af myopi gives én injektion på 2 mg i det angrebne øje.
for myopic choroidal neovascularisation, a single injection of 2 mg is given into the affected eye.

visudyne blev desuden undersøgt i to år hos 120 patienter med subfoveal koroidal neovaskularisering forårsaget af patologisk nærsynethed.
visudyne has also been studied for two years in 120 patients with subfoveal choroidal neovascularisation caused by pathologic myopia.

det har altså ikke kunnet påvises, at der er en fordel ved at bruge visudyne til okkult subfoveal koroidal neovaskularisering.
while the first study in occult disease showed some effectiveness, this was not confirmed by the second study, and the benefit of visudyne in occult subfoveal choroidal neovascularisation has not been established.

når øjnene er ankommet til laboratoriet, undersøges de omhyggeligt for defekter, herunder øget uklarhed, ridser og neovaskularisering.
the eyes, once they arrive at the laboratory, are carefully examined for defects including increased opacity, scratches, and neovascularisation.

behandling af synsnedsættelse grundet choroidal neovaskularisering (cnv) som følge af patologisk myopi (pm)
the treatment of visual impairment due to choroidal neovascularisation (cnv) secondary to pathologic myopia (pm)

til koroidal neovaskularisering forårsaget af patologisk nærsynethed rækker det hos mange patienter med kun en eller to injektioner i løbet af det første behandlingsår, mens nogle patienter kan have behov for hyppigere behandling.
for choroidal neovascularisation caused by pathological myopia, many patients may only need one or two injections during the first year of treatment, while some patients may need more frequent treatment.

binding af vegf- a til dets receptorer medfører endotelial celleproliferation og neovaskularisering samt vaskulær lækage, som alt sammen menes at bidrage til progressionen af den neovaskulære form af aldersrelateret maculadegeneration.
binding of vegf-a to its receptors leads to endothelial cell proliferation and neovascularisation, as well as vascular leakage, all of which are thought to contribute to the progression of the neovascular form of age-related macular degeneration.

disse sygdomme fører til synstab på grund af beskadigelse af nethinden, som er forårsaget af indvækst af blodkar, kaldet choroidal neovaskularisering (cnv).
these diseases lead to vision loss due to damage of the retina caused by ingrowth of blood vessels, called choroidal neovascularisation (cnv).

mens den første undersøgelse ved okkult sygdom viste nogen virkning, kunne dette ikke bekræftes i den anden undersøgelse.det har altså ikke kunnet påvises, at der er en fordel ved at bruge visudyne til okkult subfoveal koroidal neovaskularisering.
the benefit was maintained for up to 5 years.while the first study in occult disease showed some effectiveness, this was not confirmed by the second study, and the benefit of visudyne in occult subfoveal choroidal neovascularisation has not been established.

de mulige forklaringer på højere optagelse af 5-ala omfatter: nedbrudt blod-hjerne-barriere, øget neovaskularisering og overekspression af membrantransportører i gliomvæv.
explanations for higher 5-ala uptake include a disrupted blood-brain barrier, increased neo-vascularisation, and the overexpression of membrane transporters in glioma tissue.

den selektive hæmning hos dyr med pegaptanib viste sig at være ligeså effektiv til supprimering af patologisk neovaskularisering som pan-vegf hæmning, dog skånede pegaptanib den normale vaskulære struktur, hvorimod pan-vegf hæmning ikke gjorde.
the selective inhibition in animals with pegaptanib proved as effective at suppressing pathological neovascularisation as pan-vegf inhibition, however pegaptanib spared the normal vasculature whereas pan-vegf inhibition did not.