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omdannelse ved position 80, 180 og polymorf position 91, 229 blev altid påvist i sekvenser, som havde m204i-mutation, der bekræfter genotyperesistens.
conversions at position 80, 180 and polymorphic positions 91, 229 were always detected in sequences that harboured the m204i mutation that confers genotypic resistance.
qtc >500 msek. eller >60 msek. ændring fra baseline og torsade de pointes eller polymorf ventrikulær takykardi eller tegn/symptomer på alvorlig arytmi
qtc >500 msec or >60 msec change from baseline and torsade de pointes or polymorphic ventricular tachycardia or signs/symptoms of serious arrhythmia
kardiale effekter (hæmodynamisk ustabil, polymorf ventrikulær takykardi (herunder torsade de pointes)), behandles med øjeblikkelig defibrillering eller øjeblikkelig anvendelse af pacemaker.
the treatment of cardiac effects (haemodynamically unstable polymorphic ventricular tachycardia (including torsades de pointes)) is either immediate cardioversion or immediate overdrive pacing.
15 hovedmetabolitten m8 (tert- butyl hydroxynelfinavir) har in vitro antiviral aktivitet, som er lig moderstoffet, og dets dannelse er katalyseret af det polymorfe cytochrom cyp2c19.
the major oxidative metabolite, m8 (tert-butyl hydroxy nelfinavir), has in vitro antiviral activity equal to the parent drug and its formation is catalysed by the polymorphic cytochrome cyp2c19.