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the world health organization (who) has proclaimed coronavirus disease 2019 (covid-19) as a pandemic and a major public health emergency [1]. the illness ranges from asymptomatic or mild infection to acute respiratory distress syndrome (ards) with high mortality [1, 2]. presentations include fever, coughing, dyspnea, watery diarrhea, myalgia, severe lymphopenia, prolonged coagulation profiles, cardiac disease and sudden death [2, 3]. coagulopathy was frequently reported in covid-19 patients, especially among those with severe disease [4, 5]. d-dimer > 1 μg/ml is one of the risk factors of mortality in patients with covid-19 [4]. administration of low molecular weight heparin among patients with markedly elevated d-dimer level is significantly associated with better 28-day survival [6]. many autopsies of covid-19 patients showed thrombosis in small pulmonary vessels [7]. some retrospective studies found a high incidence of pe in covid-19 patients [8, 9] acute pe interferes with both circulation and gas exchange. right ventricular (rv) failure due to acute pressure overload is considered the primary cause of death in severe pe [10, 11]. d‐dimer is an indirect marker of fibrinolysis and fibrin turnover [12]. d‐dimer is a soluble fibrin degradation product that results from degradation of vascular thrombi through the fibrinolytic mechanism [12]. d‐dimer is used as a marker of activation of coagulation and fibrinolysis in a number of clinical scenarios. d-dimer test is used for exclusion of the diagnosis of venous thromboembolism (vte) and is used routinely for this indication [12].
الارتباط بين مستوى d dimer وتطور الانسداد الرئوي في المرضى الذين يعانون من فيروس كورونا الرئوي 19
Last Update: 2022-11-04
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