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Last Update: 2017-04-26
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these isolates expressed a median fold change in ec50 relative to wildtype of 12.0 for atazanavir.
disse isolater udtrykte en gennemsnitlig ændring i ec50 i forhold til vildtype på 12, 0 for atazanavir.
increasing baseline darunavir fold change in ec50 (fc) was associated with decreasing virologic response.
stigende ec50-værdi over for darunavir (målt som fc - fold change) ved baseline var associeret med faldende virologisk respons.
in the case of the q148-mutation viruses, increasing dolutegravir fold change is seen as the number of secondary mutations increase.
i tilfælde af virus med q148-mutation er der set en stigende dolutegravir-fc, efterhånden som antallet af sekundære mutationer stiger.
two mutations conferred a 7 to 8 fold change in abacavir susceptibility and combinations of three mutations were required to confer more than an 8 fold change in susceptibility.
to mutationer gav en ændring på 7 til 8 gange i følsomheden over for abacavir, og kombinationer af tre mutationer var nødvendige for at give mere end en 8 gange ændring i følsomhed.
the 16 viruses that displayed 20-fold change in susceptibility all contained mutations at positions 10, 54, 63 plus 82 and/or 84.
de 16 virus, der udviste 20 gange forskel i følsomhed havde alle mutationer ved codon 10, 54, 63 og 82 og/eller 84.
isolates with baseline fold change of 0 to 3 are considered susceptible; isolates with 3 to 10 fold changes have decreased susceptibility; isolates with 10 fold changes are resistant.
ved en baselineændring på 0-3 fold betragtes isolatet som " følsomt ".isolater med ændringer på 3-10 fold har nedsat følsomhed, mens isolater med ændringer på 10 fold er resistente.
traditional ic50/ic90 fold-change was not a useful parameter to measure phenotypic resistance, as those values were sometimes unchanged despite significantly reduced sensitivity.
traditionel ic50/ic90 gange ændring er ikke en nyttig parameter til at måle fænotypisk resistens, da disse værdier nogle gange er uændret til trods for signifikant reduceret følsomhed.
during in vitro abacavir selection the m184v mutation occurred first and resulted in about a 2 fold increase in ic50, below the abacavir clinical cut-off of 4.5 fold change.
under abacavir-selektionen in vitro opstod m184v-mutationen først og resulterede i en fordobling af ic50, under den kliniske skæring for abacavir, som er en fc på 4,5.
in general, susceptibility to atazanavir was retained (83% of isolates displayed 2.5 fold change in ec50) among isolates resistant to no more than 2 protease inhibitors.
almindeligvis blev følsomheden over for atazanavir opretholdt (83% af isolaterne udviste 2, 5 gange ændring i ec50) blandt isolater, der var resistente over for maksimalt 2 proteasehæmmere.
2.9 fold, respectively; the auc of the active metabolite of ulipristal acetate increased 1.5 fold while the cmax of the active metabolite decreased (0.52 fold change).
auc for ulipristalacetats aktive metabolit steg 1,5 gange, mens cmax for den aktive metabolit faldt (ændring på 0,52 gange).
53 -641-fold change in ec50 values) from 9 hiv-1 strains harbouring multiple pi resistance-associated mutations showed that a minimum of 8 darunavir in vitro selected mutations were required in the hiv-1 protease to render a virus resistant (fold change [ fc ] 10) to darunavir.
53-641-fold ændring i ec50-værdier) fra 9 hiv-1 stammer, der indeholdt multiple pi-resistensassocierede mutationer viste, at der krævedes mindst 8 darunavirmutationer, der blev selekteret in vitro, i hiv-1 proteasen for at gøre en virus resistent (foldændring 10) over for darunavir.