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defibrotide also inhibits the expression of heparanase contributing to extracellular matrix integrity and thereby tissue homeostasis.
defibrotide jinibixxi wkoll l- espressjoni ta' eparanase li jikkontribwixxi għall-integrità tal-matriċi ekstraċellulari u b'hekk għall- omostażi tat-tessut.
for the treatment of adults with diabetes mellitus who require insulin for the maintenance of normal glucose homeostasis.
għall-kura ta' adulti li għandhom id-dijabete mellitus li għandhom bżonn l-insulina għall-manteniment omeostatiku ta' livelli normali ta’ glukożju.
for the treatment of adults and children with diabetes mellitus who require insulin for the maintenance of normal glucose homeostasis.
għall- kura ta 'adulti u tfal li għandhom id- dijabete mellitus li għandhom bżonn l- insulina għall - manteniment omeostatiku ta' livelli normali ta ’ glucose.
humalog basal is indicated for the treatment of patients with diabetes mellitus who require insulin for the maintenance of normal glucose homeostasis.
humalog basal huwa indikat għall- kura ta 'pazjenti li għandhom id- dijabete mellitus li għandhom bżonn l- insulina għall- manteniment omeostatiku ta' livelli normali ta ’ glucose.
linagliptin glucose-dependently increases insulin secretion and lowers glucagon secretion thus resulting in an overall improvement in the glucose homeostasis.
linagliptin iżid, b’mod dipendenti fuq il-glucose, it-tnixxija tal-insulina u jnaqqas it-tnixxija ta’ glucagon, u b’hekk jirriżulta f’titjib globali fl-omeostażi tal-glucose.
the ubiquitin-proteasome pathway plays an essential role in regulating the turnover of specific proteins, thereby maintaining homeostasis within cells.
il- passaġġ proteasome ubikwitinat għandu sehem essenzjali biex jirregola l- ipproċessar ta 'proteini speċifiċi, u b' dan il- mod jinżamm l- omeostażi fiċ- ċelluli.
improvement in homeostasis model assessment for beta cell function (homa beta-cell) has been observed in clinical studies with dapagliflozin.
Ġie osservat titjib fil-mudell omeostatiku ta’ valutazzjoni għall-funzjoni taċ-ċelluli beta (homa beta-cell) fi studji kliniċi b’dapagliflozin.
improvement of surrogate markers of beta cell function including homeostasis model assessment-β (homa-β) was noted.
kien innotat titjib fil-markaturi sostituti tal-funzjoni taċ-ċelluli beta li jinkludu l-mudell omeostatiku ta' assessjar (homa-β - homeostasis model assessment-b).
clinical studies with dulaglutide have indicated enhanced beta-cell function as measured by homeostasis model assessment (homa2-%b).
studji kliniċi bi dulaglutide indikaw funzjoni aħjar taċ-ċellula beta kif imkejla bl-istima tal-mudell tal- omeostasi (homa2-%b - by homeostasis model assessment).
praziquantel is rapidly adsorbed via the surface of the parasites and affects membrane permeability in cestodes, influencing divalent cation fluxes, particularly calcium ion homeostasis, which is thought to contribute to the rapid muscle contraction and vacuolisation.
praziquantel jiġi assorbit malajr permezz tal-wiċċ tal-parassita u jeffettwa il-permejabilità tal-membrana fi cestodes, li jinfluwenza flussi ta’ divalent cation, partikolarment calcium ion homeostasis, li hu mifhum li jikkontribwixxi għal kontrazzjoni ta’ malajr tal-muskolu u vakwolizzazjoni.
improvement of surrogate markers of beta cell function, including homa-β (homeostasis model assessment-β), proinsulin to insulin ratio, and measures of beta cell responsiveness from the frequently-sampled meal tolerance test were observed.
34 assessjar -), il- proporzjon bejn proinsulina u insulina, u l- kejl tar- respons ta - elluli beta minn kampjuni me uda frekwentament fit- test tat- tolleranza g all- ikel ew osservati.