Results for vagal translation from English to Maltese

serotonin then activates 5-ht3 receptors located on vagal afferents to initiate the vomiting reflex.
serotonin imbagħad jattiva r-riċetturi ta’ 5-ht3 li jinsabu fuq afferenti vagali biex jibda l-proċess tar-rimettar.

the most likely manifestation of overdosage would be hypotension and tachycardia; bradycardia could occur from parasympathetic (vagal) stimulation.
l- aktar manifestazzjoni probabbli ta ’ doża eċċessiva tkun presjoni baxxa u takikardja; bradikardja tista ’ sseħħ minn stimulazzjoni parasimpatetika (vegus).

between 85.3% and 89.1% of patients were taking two to three concomitant aeds with or without concurrent vagal nerve stimulation.
bejn 85.3% u 89.1% tal-pazjenti kienu qed jieħdu tnejn jew tlett aeds fl-istess ħin bi jew mingħajr stimulazzjoni tan-nervi vagali fl-istess ħin.

in addition, rare cases of transient loss of consciousness, which may be preceded by vagal symptoms, have been reported (see section 4.8).
addizzjonalment, każijiet rari ta’ telf mis-sensi għal żmien qasir, li jistgħu jkunu preċeduti minn sintomi ikkaġunati minn nervaturi tal-vagus, ġew irrapportati (ara sezzjoni 4.8).

there have been post-marketing adverse reaction reports of transient loss of consciousness including some cases associated with vagal syndrome (see sections 4.7 and 4.8).
wara li l-prodott tpoġġa fuq is-suq, kien hemm rapporti ta’ reazzjonijiet avversi ta’ telf ħafif mis-sensi u kienu jinkludu xi każijiet assoċjati mas-sindromu vagal (ara sezzjonijiet 4.7 u 4.8).

low doses of intravenous sufentanil associated with likely vagal (cholinergic) activity cause mild bradycardia and mildly reduced systemic vascular resistance without significantly lowering blood pressure (see section 4.4).
dożi baxxi ta’ sufentanil mogħti ġol-vini assoċjati mal-probabbiltà ta’ attività vagali (kolinerġika) jikkawżaw bradikardija ħafifa u reżistenza vaskulari sistematika ftit imnaqqsa mingħajr ma jbaxxu b’mod sinifikanti l-pressjoni tad-demm (ara sezzjoni 4.4).