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studies in rabbits demonstrated that the topically administered nepafenac is distributed locally from the front of the eye to the posterior segments of the eye (retina and choroid).
studier med kaniner viste, at topikalt administreret nepafenac distribueres lokalt fra forsiden af øjet til øjets posteriore segment (retina og choroidea).
pharmacodynamic effects the majority of hydrolytic conversion is in the retina/ choroid followed by the iris/ ciliary body and cornea, consistent with the degree of vascularised tissue.
farmakodynamisk virkning hovedparten af den hydrolytiske konvertering foregår i retina/ choroidea efterfulgt af iris/ corpus ciliare og hornhinden svarende til graden af vaskulariseret væv.
levels of ocular toxicity in healthy rabbits and monkeys, particularly on the retina/choroid, correlated with medicinal product dose, light dose, and time of light treatment.
graden af okulær toksicitet, særligt vedrørende retina/choroidea, hos raske kaniner og aber korrelerede med lægemiddeldosis, lysdosis og varigheden af lysbehandling.
in vitro iaa also inhibits the accumulation of c14-labelled aa in the isolated rabbit choroid plexus (shibata, m.a. et al., 1989).
også in vitro hæmmer ias akkumulationen af c14-mærket as i isoleret plexus chorioideus hos kaniner (shibata, ma. et al. 1989).
ex vivo, a single topical ocular dose of nepafenac was shown to inhibit prostaglandin synthesis in the iris/ ciliary body (85%-95%) and the retina/ choroid (55%) for up to 6 hours and 4 hours, respectively.
ex vivo blev påvist, at én enkelt topikal okulær dosering af nepafenac hæmmer prostaglandinsyntese i iris/ corpus ciliare (85% - 95%) og i retina/ choroidea (55%) i op til henholdsvis 6 og 4 timer.