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excessive coagulation activation in the microcirculatory bed plays a significant part in the pathophysiology of severe sepsis.
excessiv aktivering af koagulationen i det mikrocirkulatoriske karsystem spiller en væsentlig rolle i patofysiologien af svær sepsis.
both endothelial cell damage and prothrombotic-hypofibrinolytic state are critical factors in the pathophysiology of this disease.
både endotelcellebeskadigelse og en protrombotisk-hypofibrinolytisk tilstand er afgørende faktorer i denne sygdoms patofysiologi.
due to the different pathophysiology of arrhythymias it is essential to have a variety of different acting medicines in order to be able to treat the specific condition.
på grund af hjertearytmis forskellige patofysiologier er det nødvendigt at disponere over en række forskelligtvirkende lægemidler for at kunne behandle den pågældende lidelse.
whether the mechanism of action of avonex in ms is mediated by the same pathway as the biological effects described above is not known because the pathophysiology of ms is not well established.
hvorvidt virkningsmekanismen for avonex i ms sker via de samme kanaler som de biologiske virkninger, der er beskrevet herover, vides ikke, da patofysiologien for ms ikke er velbeskrevet.
although the pathophysiology may be the same as for sar, patients suffering from par suffer the whole year, which implies a more chronic use of the combination of loratadine and pseudoephedrine.
selv om patofysiologien kan være den samme som for sar, lider patienter, som lider af par, hele året, hvilket indebærer en mere kronisk brug af kombinationen af loratadin og pseudoephedrin.
this difference is perhaps explained by the anatomically and pathophysiology more complicated clinical setting of osteomyelitis in the diabetic foot, making differentiation of soft tissue and bone infection more difficult than in other presentations of long bone osteomyelitis.
denne forskel kan måske forklares ved den anatomiske og patofysiologisk mere komplicerede kliniske situation ved osteomyelitis i den diabetiske fod, der gør differentiering mellem infektion i bløddelsvæv og knogler mere vanskelig end ved andre former for osteomyelitis i rørknogler.
chronic idiopathic urticaria was studied as a clinical model for urticarial conditions, since the underlying pathophysiology is similar, regardless of etiology, and because chronic patients can be more easily recruited prospectively.
kronisk idiopatisk urticaria er blevet undersøgt som en klinisk model for urticarialidelser, da den patofysiologi der ligger til grund herfor er tilsvarende uanset ætiologi, og fordi kroniske patienter lettere kan rekrutteres prospektivt.
the main focus of the health theme in the rare diseases area are europe-wide studies of natural history, pathophysiology, and the development of preventive, diagnostic and therapeutic interventions.
mere specifikt ligger fokus for sundhedstemaet i forbindelse med sjældne sygdomme på europæiske undersøgelser inden for naturhistorie, patofysiologi og udvikling af forebyggelse, diagnosticering og behandling.
velaglucerase alfa supplements or replaces beta-glucocerebrosidase, the enzyme that catalyzes the hydrolysis of glucocerebroside to glucose and ceramide in the lysosome, reducing the amount of accumulated glucocerebroside and correcting the pathophysiology of gaucher disease.
velaglucerase alfa supplerer eller erstatter betaglucocerebrosidase (det enzym, der katalyserer hydrolysen af glucocerebrosid til glukose og ceramid i lysosomet), hvorved mængden af ophobet glucocerebrosid mindskes, og hvorved patofysiologien for gauchers sygdom korrigeres.
imiglucerase (recombinant macrophage targeted acid ß-glucosidase) replaces the deficient enzyme activity, hydrolysing glucosylceramide, thus correcting initial pathophysiology and preventing secondary pathology.
imiglucerase (rekombinant makrofag-målrettet ß-glucosidasesyre) erstatter den manglende enzym- aktivitet, hydrolyserer glucosylceramid og korrigerer dermed den indledende patofysiologi og forhindrer sekundær patologi.
86 latency/latency period 37,73 lead chromate pigment exposure, lung cancer and 63-5 liver cancer, vinyl chloride exposure and 16,17,20,36 epidemiology (cont.): definition 23 in practice 45-65 excess rate (rate difference) 77-8 expected vs observed cases 12-13 exposed and nonexposed persons, lungcancer 31-2 chromate pigment exposure and 63-5 smoking and 31-2,35-6,64-5 vinyl chloride exposure and 23,36 lung disease, welding and 24 selection 27- & exposure to substances in casecontrol studies 21,24-5 in diseased vs non-diseased 16 in follow-up studies 25-6 information/data on of individuals 30 obtaining 24-5 monitoring 7,43 time factors 37 see also substances matching 74 in casecontrol studies 20-1 confounding avoided by 36 metal-can factory, 'flu' epidemic in 45-9 misclassification (into disease or exposure category) 34-5,74 non-differential/random 35 monitoring 42-3 of exposure 7,43 mortality ratio proportional 40-2,76-7 problems with 41 standardized 40,76,79, 80-1 in follow-up studies 28 see also death mortality studies 40-2 nasal cancer, cabinet makers and 23-4 fertility problem, l,2-dibromo-3-dichloropropane exposure causing 16,20,23,49-53 flax-mill, byssinosis and chronic bronchitis in 59-60 'flu' epidemic, occupational 45-9 follow-up 71 time 29 follow-up study (cohort study) 25-30, 71-2 historical 29-30 ongoing 26-7 study base in 83 frequency of disease, measures 74 gender as a confounder 36 goal, defining the 3 health problem, see problem health worker selection and the healthy worker effect 28-9 objective data 33 observed vs expected cases 12-13 occurrence, see prevalence odds ratio 74-5 in casecontrol studies 22 office of population census and surveys, information available from 6,7 oligozoospermia in pesticide factory workers 50,51 incidence (rate) 13,72-3 prevalence and, relationship 39 infertility, 1,2-dibromo-3-dichloropropane exposure and 16,20,23,49-53 pathophysiology, exposure studies and knowledge of 26 pesticide factory workers, infertility in 16,20,23,49-53 plastic workshop of metal-can factory, 'flu' epidemic associated information biased 34-5 exposure, obtaining 24-5 useful and necessary 14-15 with 47-8 population, reference 78 power plant, cardiovascular disease in interaction 73 isocyanate and chronic bronchitis 38-9 60-2
sygdomstilfælde forventede 24 observerede 24 relativ risiko (rr) 26, 43, 114 risiko 114 „risiko-personer" 44 risikoindikatorer 50 tiden 58 tværsnitsundersøgelse 60, 119 undersøgelsesgruppe (studiebase) 32, 33, 119 videnskabsetiske komiteer 69 vinylchlorid 25 ætiologi (årsagssammnehænge) 119 ætiologisk fraktion 119 sammenligningsgyldighed 54 selektion 53 ^ smr (standardiseret mortalitets ratio) 63, 115 snow, john 51 standardiseret mortalitets ratio (smr) 63, 115 standardisering 114 stratificeret analyse 117 stratifikation 117 studiebase (undersøgelsesgruppe) 33, 118 årsager (ætiologiske faktorer) 121 årsagssammenhænge 50
