Hai cercato la traduzione di train short terminate at ara da Inglese a Hindi (indiano)

short terminate at ambala
ट्रेन गंतव्य के लिए समाप्त हो गई

they were travelling in a train which was to terminate at jammu
वे एक ट्रेन में यात्रा कर रहे थे जो जम्मू तक जाने वाली थी

the scheme will terminate at the end of the ixth plan on 31 . 3 . 2002 .
यह योजना 31 . 3 . 2002 को 9वीं योजना के समाप्त होने पर समाप्त हो जाएगी ।

such funds which are for a fixed term and terminate at the end of the period .
ऐसी निधियां जो एक नियत समय के लिए हों तथा उस कालावधि के पश्चात समाप्त हो जाती हों ।

it terminates at the posterior end of body , after forming an anal ganglion .
गुद् गुच्छिका के निर्माण के बाद शरीर के बाहरी अंत पर वह समाप्त होता है ।

the chiantar glacier flows for 32 km to the west from a high peak northeast of the darkot pass and terminates at 3 , 658 m 1959 .
चियान्तर हिमनद , दारकोट दर्रे के उत्तर - पूर्व स्थित एक ऊंचे पर्वत शिखर से पश्चिम की ओर 32 किमी . तक प्रवाहित होने के बाद 3 , 658 मी . 1959 की ऊंचाई पर समाप्त होता है ।

the third one , the gangstung glacier , is only 6 km long and it terminates at 4 , 350 m elevation 1963 .
एक तीसरा गंगत्संगु हिमनद है जो मात्र 6 किमी . लंबा है तथा 4 , 350 मी . की ऊंचाई पर 1963 समाप्त होता है ।

the khumbu glacier . on the south of the everest is 20 km long and terminates at an elevation of 4 , 900 m 1963 in an enormous terminal moraine 460m high and composed of at least 12 ridges .
एवरेस्ट के दक्षिण से उद्गमित खुम्बू हिमनद 20 किमी . लंबा है और इसका अंत 4 , 900 मी . 1963 की उचांई पर होता है , जो 460 मी . ऊंचे तथा कम - से - कम 12 भित्तियों वाले अंतस्थ हिमोढ़ है ।

duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long proteins can have different localization, stability and function. “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.
duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long prote “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.