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this morphology is created by the viral spike peplomers, which are proteins on the surface of the virus.
mofolojia hii imeundwa na miiba ya glikoprotini kwenye virusi, ambayo ni protini kwenye uso wa virusi.
the lipid bilayer envelope, membrane proteins, and nucleocapsid protect the virus when it is outside the host cell.
mfuniko wa tabaka mbili za lipidi, protini za utandao, na nucleocapsid hulinda virusi wakati viko nje ya seli mama.
analyzing those binding sites provide the reasonable project of developing effective antiviral drug against covid-19 proteins.
kuchambua maeneo hayo ya kuunganisha hutoa mradi mzuri wa kutengeneza dawa bora ya kuzuia virusi itakayotoa protini za covid-19.
more pathogenic cov strains might also evolve by recombination, leading to the acquisition of novel proteins or protein features for host adaptation.
sampuli zaidi za kusababisha ugonjwa za cov zinaweza kukua kupitia uunganishaji tena, na kusababisha kupatikana kwa protini mpya au sifa za protini kwa ajili ya kuzoeleka kwenye mbebaji.
there, the m proteins direct most protein-protein interactions required for assembly of viruses following its binding to the nucleocapsid.
hapo, prorini ya m huelekeza miingiliano baina ya protini na protini inayohitajika inayohitajika kwa mkusanyiko wa virusi kufuatia kunatwa kwake kwenye nucleocapsidi.
the divergence of these host proteins between humans and natural reservoir hosts of hcovs such as bats, dromedary camels and rodents might constitute a barrier to interspecies transmission.
tofauti ya protini hizi za wanyama wabebaji kati ya binadamu na wabebaji asilia wahifadhi wa virusi vya hcov kama vile popo, ngamia warefu zaidi na wanyama wagugunaji huenda ukawa kikwazo kwa usambazaji baina ya viumbe.
a number of the nonstructural proteins coalesce to form a multi-protein replicase-transcriptase complex (rtc).
idadi ya protini zisizokuwa na muundo hushirikiana ili kuunda multi-protein replicase-transcriptase complex (rtc).
around 24 january 2020 in australia, the university of queensland announced that it is investigating the potential of a molecular clamp vaccine that would genetically modify viral proteins in order to stimulate an immune reaction.
mnamo 24 januari 2020 nchini australia, chuo kikuu cha queensland kilitangaza kwamba kinafanya uchunguzi wa uwezekano wa chanjo ya kunata molekuli inayoweza kubadilisha kijenetiki protini za virusi ili kuchochea kinga ya mwili.
they found that the complex, which had open and closed conformations, was assembled as a dimer and the ace2-b0at1 complex can bind two s proteins, which provides evidence for cov recognition and infection.
waligundua kwamba uchangamano huo, uliokuwa na pande wazi na funge za kuendana, ulikusanywa kama molekuli mbili na uchangamano wa ace2-b0at1 unaweza kuunganisha protini mbili za s, jambo linalotoa ushahidi wa utambuzi na maambukizi ya cov.