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genotypisk
genotypical

genotypisk koefficient
genotypical coefficient

genotypisk resistens:
genotypic resistance:

score ifm. genotypisk følsomhed* < 2
genotypic susceptibility score* <2

genotypisk sensitivitetsscore ved baseline kunne ikke skaffes for alle patienter.
genotypic sensitivity scores were not obtained for all patients at baseline.

ob var valgt ud fra patienternes tidligere behandlingsanamnese og genotypisk og fænotypisk viral resistensbestemmelse ved baseline.
the obt was selected on the basis of the subject’s prior treatment history and baseline genotypic and phenotypic viral resistance measurements.

genotypisk resistens:resistensprofilen over for virus hos behandlingserfarne forsøgspersoner er endnu ikke fuldt karakteriseret.
12 genotypic resistance: the resistance profile of virus from treatment-experienced subjects has not yet been fully characterized.

et bedre respons er observeret hos patienter med genotypisk sensitivitets-score (gss) 0.
higher response rates were observed in patients with genotypic sensitivity score (gss) 0.

i pivotalstudiet var graden af genotypisk verificeret telbivudin- resistens 2, 7% efter 48 uger.
the rate of genotypically confirmed telbivudine resistance in the pivotal study at 48 weeks was 2.7%.

før randomisering blev obt valgt af investigator på baggrund af patientens behandlingsanamnese, samt testning af genotypisk og fænotypisk virusresistens ved baseline.
prior to randomization, obt were selected by the investigator based on the patient's prior treatment history, as well as baseline genotypic and phenotypic viral resistance testing.

genotypisk resistens over for efavirenz, overvejende k103n-mutationen, forekom hyppigst (tabel 5) .
genotypic resistance to efavirenz, predominantly the k103n mutation, occurred most frequently (table 5) .

sofosbuvir er en pan-genotypisk hæmmer af hcv ns5b rna-afhængig rna-polymerase, som er essentiel for viral replikation.
sofosbuvir is a pan-genotypic inhibitor of the hcv ns5b rna-dependent rna polymerase, which is essential for viral replication.

18 indgivet sammen med lavdosis ritonavir viser et bedre behandlingsrespons efter 48 uger, når den optimerede baggrundsbehandling indeholder genotypisk tilgængelige antiretrovirale midler (fx enfuvirtid) .
resist data also demonstrate that aptivus co-administered with low dose ritonavir exhibits a better treatment response at 48 weeks when the obr contains genotypically available antiretroviral agents (eg enfuvirtide) .

nogle genotypisk resistens data indikerer, at hos de fleste patienter resistente for nnrti, findes virale anlæg for kryds- resistens til de andre nnrti’ er.
some genotypic resistance data indicate that in most patients failing nnrti viral strains express cross-resistance to the other nnrtis.

lægemiddelresistens måles som ændringen i viral følsomhed overfor lægemidlet ved dyrkning (" fænotypisk resistens ") eller i protease aminosyresekvensen (" genotypisk resistens ") .
drug resistance is measured as the change in viral susceptibility to drug in culture (="phenotypic resistance ") or in protease amino acid sequence (="genotypic resistance ") .

der blev gennem den 48 ugers behandlingstid foretaget to typer analyser baseret på genotypiske mutationer ved baseline.
through 48 weeks of treatment, two types of analyses were performed based on baseline genotypic mutations.