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efflux viscosimeter
udløbsviskosimeter

· drug efflux pumps
· udstrømningspumper.

ambrisentan is a substrate for pgp-mediated efflux.
ambrisentan er et substrat for p-gp-medieret effluks.

eluxadoline is a substrate of the hepatic efflux transporter mrp2.
eluxadolin er substrat for levereffluxtransportøren mrp2.

edoxaban is a substrate for the efflux transporter p-gp.
edoxaban er et substrat for effluks-transportproteinet p-gp.

maraviroc is a substrate for the efflux transporter p-glycoprotein.
maraviroc er et substrat for efflux-transporteren p- glycoprotein.

inhibition of the transporters that efflux chloroquine from the parasite food vacuole
hæmning af transportørerne, som får chloroquin til at strømme ud af parasittens fødevakuole.

dabigatran etexilate is a substrate for the efflux transporter p-gp.
dabigatranetexilat er et substrat for efflukstransporteren p-gp.

a rapid uptake of everolimus in the brain followed by a slow efflux.
et hurtigt optag af everolimus i hjernen efterfulgt af langsom effluks

resistance may also be mediated by bacterial impermeability or by bacterial drug efflux pumps.
resistens kan også medieres ved bakteriel uigennemtrængelighed eller ved bakterielle udstrømningspumper i stoffer.

moxifloxacin is a poor substrate for active efflux mechanisms in gram-positive organisms.
moxifloxacin er et dårligt substrat for aktiv effluks- mekanismer i grampositive organismer.

decreased susceptibility in acinetobacter baumannii has been attributed to the overexpression of the adeabc efflux pump.
nedsat følsomhed over for acinetobacter baumannii er blevet tilskrevet overekspression af adeabc efflukspumpen.

ritonavir may increase digoxin levels due to modification of p-glycoprotein mediated digoxin efflux.
ritonavir kan øge digoxinkoncentrationen på grund af modificering af p- glycoproteinmedieret digoxinefflux.

colchicine is a substrate of cyp3a4 and p-gp (an intestinal efflux transporter).
colchicin er cyp3a4 og p-gp substrat (en intestinal efflukstransporter).

inhibitors of p-gp may decrease the efflux of sirolimus from intestinal cells and increase sirolimus levels.
p- gp hæmmere kan nedsætte udstrømningen af sirolimus fra tarmcellerne og øge sirolimusniveauet.

this non-target specific efflux mechanism has also been demonstrated to reduce the in vitro activity of streptogramin a.
det er også vist, at denne non- specifikke efflux mekanisme reducerer in vitro-aktiviteten af streptogramin a.

calcium folinate and folate antagonists share the same membrane transport carrier and compete for transport into cells, stimulating folate antagonist efflux.
calciumfolinat og folatantagonister bruger samme membrantransporter og konkurrerer om transport ind i cellerne, sådan at folatantagonist efflux stimuleres.

decreased susceptibility in both groups has been attributed to the overexpression of the non-specific acrab multi-drug efflux pump.
den nedsatte følsomhed i begge grupper er blevet tilskrevet overekspression af den ikke-specifikke acrab-efflukspumpe til flere lægemidler.

cyp 3a4 inhibition by tipranavir/r and p-gp (an intestinal efflux transporter) inhibition by clarithromycin.
tipranavir/r hæmmer cyp3a4, og clarithromycin hæmmer p-gp (p- glykoprotein, en intestinal efflukstransporter).

one involves mutations in ribosomal protein l3, the other is a non- specific efflux mechanism (abc transporter vgaav).
den ene mekaniske skyldes en mutation i det ribosomale protein l3, mens den anden er en non- specifik efflux- mekanisme (abc transporter vgaav).