Je was op zoek naar: esterase (Engels - Maltees)

urine leukocyte esterase positive, n (%)
pożittiv għal esterase lewkoċiti fl-awrina, n (%)

if anticholine esterase activity is detected a test for response to reactivating agents should be considered
jekk tinstab attività anticholine esterase għandu jitqies li jsir test għar-reazzjoni għall-aġenti ta' riattivazzjoni

1 unit of conestat alfa activity is defined as the equivalent of c1 esterase inhibiting activity present in 1 ml of pooled normal plasma.
unità waħda ta’ attività ta’ conestat alfa hija definita bħala l-ekwivalenti tal-attività inibitorja ta’ esterase c1 preżenti f’1 ml ta’ plasma normali miġbura.

the cleavage of the prodrug dabigatran etexilate by esterase-catalysed hydrolysis to the active principle dabigatran is the predominant metabolic reaction.
il- qsim tal- prodrug dabigatran etexilate permezz ta ’ idrolisi kkatalizzata minn esterase

after oral administration, dabigatran etexilate is rapidly absorbed and converted to dabigatran by esterase-catalysed hydrolysis in plasma and in the liver.
wara l- għoti mill-ħalq, dabigatran etexilate ikun assorbit malajr u jinbidel għal dabigatran permezz ta’ idrolisi kkatalizzata minn esterase fil-plażma u fil-fwied.

hae (an autosomal dominant disease) is caused by an absence or dysfunction of c1-esterase- inhibitor.
l-hae (marda awtosomali dominanti) hija kkawżata minn nuqqas jew disfunzjoni ta’ l-inibitur esterase c1.

finally, the committee was of the opinion that nte (neuropathy target esterase) inhibition by fosthiazate and its isomers had not been adequately assessed.
finalment il-kumitat kien ta' l-opinjoni illi l-nte (neuropathy target esterase) inibizzjoni minn fosthiazate u l-iżomeri tiegħu ma kienux ġew stmati adegwatament.

coagulation factor v, clotting factor viii, 2-macroglobulin, and c1-esterase inhibitor are only slightly reduced and normalise within 1 to 2 days.
il-fattur v tat-tagħqid tad-demm , fattur viii tat-tagħqid tad-demm , 2-makroglobulina, u l- inibitur ta’ c1-esterase huma biss kemmxejn imnaqqsa u jinnormalizzaw fi żmien jum sa jumejn.

these patients presented with abdominal pain (with or without nausea and vomiting); in some cases there was no prior facial angioedema and c-1 esterase levels were normal.
dawn il- pazjenti ppreżentaw rwieħom b’ uġigħ addominali (bi jew mingħajr tqalliegħ u rimettar); f’ xi wħud mill - każijiet ma ’ kienx hemm anġjoedema tal- wiċċ qabel u l- livelli ta ’ c- 1 esterase kienu normali.

in vitro and in vivo, clopidogrel is metabolised according to two main metabolic pathways: one mediated by esterases and leading to hydrolysis into its inactive carboxylic acid derivative (85% of circulating metabolites), and one mediated by multiple cytochromes p450.
in vitro u in vivo, clopidogrel jiġi mmetabolizzat permezz ta' żewġ sensiliet ta' reazzjonijiet metaboliċi: waħda bl-intervent ta' esterases u li twassal għall- idroliżi fid-derivattiv inattiv tiegħu carboxylic acid (85% tal-metaboliti fiċ-ċirkulazzjoni), u waħda bl- intervent ta' ċitokromi p450 multipli.