Je was op zoek naar: triphosphate (Engels - Maltees)

half-life of clofarabine triphosphate
β half- life ta ’ clofarabine half- life ta ’ clofarabine triphosphate tneħħija sistematika tneħħija renali

sodium triphosphate (sodium tripolyphosphates)
trifosfat tas-sodju (tripolifosfat tas-sodju)

emtricitabine is phosphorylated by cellular enzymes to form emtricitabine triphosphate.
emtricitabine jiġi fosforilat permezz ta’ enzimi ċellulari biex jifforma emtricitabine triphosphate.

it is metabolised intracellularly to the active moiety, lamivudine 5’- triphosphate.
jiġi mmetabolizzat fiċ-ċelluli għal komponent attiv, lamivudine 5'- triphosphate.

it is metabolised intracellularly to the active moiety, lamivudine 5’ - triphosphate.
jiġi metabolizzat fiċ- ċelluli għal komponent attiv, lamivudine 5 '- triphosphate.

ribonucleotide reductase inhibition with reduction of cellular deoxynucleotide triphosphate (dntp) pools.
inibizzjoni ta’ ribonucleotide reductase bi tnaqqis ta’ għadajjar ta’ cellular deoxynucleotide triphosphate (dntp).

emtricitabine and tenofovir are phosphorylated by cellular enzymes to form emtricitabine triphosphate and tenofovir diphosphate, respectively.
emtricitabine u tenofovir huma fosforilati minn enzimi ċellulari sabiex jifformaw emtricitabine triphosphate u tenofovir diphosphate, rispettivament.

abacavir is metabolised intracellularly to the active moiety, carbovir 5’- triphosphate (tp).
abacavir jiġi mmetaboliżżat fiċ-ċelloli għal sustanza attiva, carbovir 5’-triphosphate (tp).

the monophosphate metabolite is subsequently converted to the active 5’ -triphosphate form, ara-gtp.
il- prodott tal- metaboliżmu monophosphate imbagħad jinbidel fil- forma attiva ta '5' triphosphate, ara- gtp.

emtricitabine triphosphate competitively inhibits hiv-1 reverse transcriptase (rt), resulting in dna chain termination.
emtricitabine triphosphate jimpedixxi b’mod kompetittiv ir-reverse transcriptase tal-hiv-1, li jirriżulta f’terminazzjoni tal-katina tad-dna.

incorporation of telbivudine-5'-triphosphate into viral dna causes dna chain termination, resulting in inhibition of hbv replication.
meta telbivudine- 5 ’ - triphosphatase jiġi nkorporat fid- dna tal- virus, il- katina tad- dna tiġi terminata, u b’ hekk ir - replikazzjoni ta ’ hbv tieqaf.

phosphates (excluding calcium hydrogenorthophosphate and mono- or disodium phosphate); polyphosphates (excluding sodium triphosphate)
fosfati (minbarra idroġenu tal-kalċju jew tofosfat u fosfati ta mono- jew disodju); polifosfati (minbarra trifosfat tas-sodju)

by contrast, telbivudine-5'-triphosphate at concentrations up to 100 m did not inhibit cellular dna polymerases , , or .
b’kuntrast, konċentrazzjonijiet ta’ telbivudine-5’-triphosphate sa 100 m ma wasslux għall- inibizzjoni ta’ dna polymerases ċellulari , , jew .

both medicinal products are metabolised intracellularly to their active moieties, lamivudine 5’-triphosphate (tp) and zidovudine 5’-tp respectively.
iż- żewġ prodotti mediċinali huma metabolizzati ġoċ-ċelluli għall-mojetijiet attivi tagħhom, lamivudine 5’– triphosphate (tp) u zidovudine 5’-tp rispettivament.

telbivudine-5'-triphosphate inhibits hbv dna polymerase (reverse transcriptase) by competing with the natural substrate, thymidine 5'-triphosphate.
telbivudine- 5 ’ - triphosphate jinibixxi hbv dna polymerase (reverse transcriptase) billi jikkompeti mas- sustrat naturali, thymidine- 5 ’ - triphosphate.

abacavir and lamivudine triphosphates show significantly less affinity for host cell dna polymerases.
abacavir u lamivudine triphosphates juru affinita’ sostanzjalment inqas għal polymerases tad-dna taċ-ċellula reċipjenta.