Je was op zoek naar: upregulated (Engels - Maltees)

in disease conditions such as pah, the local et system is upregulated and is involved in vascular hypertrophy and in organ damage.
f’kondizzjonijiet ta’ mard bħal pah, is-sistema lokali ta’ et tiħrax u hija nvoluta f’ipertrofija vaskulari u fi ħsara lill-organi.

endogenous kgf is an epithelial cell specific growth factor which is produced by mesenchymal cells and is naturally upregulated in response to epithelial tissue injury.
kgf endoġenu huwa fattur tat-tkabbir speċifiku taċ-ċelluli tal- epitilju li huwa magħmul minn ċelluli mesenkimali, u l-ħelsien tiegħu jiżdied b’mod naturali b’rispons għall-ħsara fit-tessut tal-epitilju.

however, in the presence of pro-inflammatory cytokines, vcam-1 is upregulated on endothelial cells and possibly on glial cells near the sites of inflammation.
iżda fil-preżenza ta’ ċitokini li jippromwovu l-infjammazjoni, tiġi attivata aktar mis-soltu l-vcam-1 fuq iċ-ċelluli endotiljali u possibilment fuq iċ-ċelluli glijali qrib il-postijiet ta’ infjammazzjoni.

as the bile acid pool becomes depleted, the hepatic enzyme, cholesterol 7-α -hydroxylase, is upregulated, which increases the conversion of cholesterol to bile acids.
hekk kif l- ammont ta ’ aċidu tal- bili jonqos sew, l- enżima epatika, cholesterol 7- α- hydroxylase, jiżdied, li jżid il- konverżjoni tal- kolesterol f’ aċidi tal- bili.

everolimus is a selective mtor (mammalian target of rapamycin) inhibitor. mtor is a key serine-threonine kinase, the activity of which is known to be upregulated in a number of human cancers.
everolimus huwa impeditur selettiv ta’ mtor (mira mammifera ta’ rapamycin). mtor huwa serine- threonine kinase prinċipali, li l-attività tiegħu hi magħrufa li tiġi supraregolata f’għadd ta’ kanċer fil- bniedem.