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whether citizen of india
Laatste Update: 2023-07-04
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speciality of india
specialty of india
Laatste Update: 2017-07-13
Gebruiksfrequentie: 2
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farmer son of india
Laatste Update: 2020-12-24
Gebruiksfrequentie: 1
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who is president of india
who is the president of india
Laatste Update: 2022-08-10
Gebruiksfrequentie: 2
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the farmer of india not rich
the ashok' brothers was not thief
Laatste Update: 2021-08-26
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the variety and unity of india
the variety and unity of india
Laatste Update: 2022-02-01
Gebruiksfrequentie: 1
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name the longest river of india?
longest river were named of india?
Laatste Update: 2016-08-01
Gebruiksfrequentie: 1
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role play on narendra modi pm of india
narendra modi on the role of pm of india
Laatste Update: 2022-04-23
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a script on freedom struggle of india
a script on independence struggle of india
Laatste Update: 2018-08-01
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essay on 10 birds of india in hindi
essay on 10 birds of india in hindi
Laatste Update: 2017-07-01
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president of india nikle mere ghar ke samne se
president of india nikle mere ghar ke samne se
Laatste Update: 2021-06-27
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assam is situated in the north east part of india
Laatste Update: 2023-12-16
Gebruiksfrequentie: 1
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delhi is tha capital of india isn't delhi
Laatste Update: 2024-03-03
Gebruiksfrequentie: 1
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mera 592210110001171ka bank of india ka account bhula gaya h
my account has been deleted.
Laatste Update: 2019-01-16
Gebruiksfrequentie: 1
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maine meeting attend ki exporters ki inaugrated bank of india ki trf se noida mai
maine meeting attend ki exporters ki inaugurated bank of india ki trf se noida mai
Laatste Update: 2022-05-05
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let us remember the golden heritage of our country and feel proud to be a part of india
malayalam essay on indian flag
Laatste Update: 2021-08-15
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word problem the population of india in 1995 was 96 32 80 529. write the population in words
word problem the population of india in 1995 was 96 32 80 529. write the population in words
Laatste Update: 2021-05-14
Gebruiksfrequentie: 1
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your account is in-principle eligible under new restructuring scheme. please contact your branch for further details/formalities. union bank of india
Laatste Update: 2021-05-26
Gebruiksfrequentie: 2
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hindi is the official language of india it is also regarded as the national language of the country it is spoken by a large number of people of india hindi is a simple and rich language i can speak hindi very well
Laatste Update: 2023-06-08
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duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long prote “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.
duchenne muscular dystrophy, a severe type of muscle weakness that usually begins at an early age and worsens quickly, may soon have a new strategy of treatment through genetic regulation. there is no known cure for duchenne muscular dystrophy. treatments usually aim to control symptoms to improve quality of life. sandeep eswarappa, assistant professor indian institute of science (iisc), bengaluru one of the 21 recipients of this year’s swarnajayanti fellowship of the department of science and technology (dst), government of india proposes to supress the disease-causing premature stop codon or the genetic process that initiates these diseases. he is trying to bring about the suppression through translational readthrough, a gene regulatory principle found in humans, yeasts, bacteria and drosophila which takes place with the variation of the genetic code. prof. sandeep’s group has been developing strategies to induce translational readthrough across genetic diseases caused by non-sense mutations --a change in dna that causes a protein to terminate or end its translation earlier than expected. they were successful in achieving this in vitro in case of thalassemia and are working on other disease models. this research work has been published in the scientific journal ‘biochemistry’ recently. with the swarna jayanti fellowship, they will extend it to duchenne muscular dystrophy. if successful, this project may lead to novel therapeutics for the treatment of genetic diseases like thalassemia, duchenne muscular dystrophy, haemophilia. in case of any protein formation genetic information present in the genome is first transcribed into an mrna, which in turn is translated into a protein. protein synthesis or translation is executed by macromolecular machinery called ribosomes. ribosomes start this process at a specific location on an mrna called ‘start codon’ and terminate at a stop signal called ‘stop codon’. in case of diseases with nonsense mutations, such mutations result in premature stop signal in mrna often resulting in non-functional truncated protein. prof. sandeep eswarappa’s laboratory at iisc has shown that in certain mrnas, under certain conditions, translating ribosomes misread the stop signal and continue till they encounter another stop signal. in this translational readthrough process, a longer protein is synthesized with an extension. this extension might change the properties of the protein. the experiments carried out by his group have revealed that such long proteins can have different localization, stability and function. “the knowledge we have already gained from our experiment have opened an unexpected avenue to treat genetic diseases caused by non-sense mutations like duchenne muscular dystrophy, haemophilia and so on,” said prof. sandeep.
Laatste Update: 2021-03-15
Gebruiksfrequentie: 1
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