Je was op zoek naar: autonomic (Russisch - Engels)

autonomic innervation.
autonomic innervation.

the autonomic nervous system and hypertension.
the autonomic nervous system and hypertension.

freud and dreams autonomic nervous system
he may be reelected but no longer than for two terms.

shlyakhto ev, konradi ao, tsyrlin va. the autonomic nervous system and hypertension.
the autonomic nervous system and hypertension.

the machines do not even need to be autonomic. consider the predator drones, currently in use.
the machines do not even need to be autonomic. consider the predator drones, currently in use.

== effects ==when the receptor is activated to open the ion channel by agonists, the following effects are observed:* cns: nausea and vomiting center in brain stem, anxiety, seizure propensity* pns: neuronal excitation (in autonomic, nociceptive neurons), emesis== agonists ==agonists for the receptor include:* 2-methyl-5-ht* ethanol* alpha-methyltryptamine* ibogaine* bufotenin* varenicline* chlorophenylbiguanide* quipazine* rs-56812: potent and selective 5-ht3 partial agonist, 1000x selectivity over other serotonin receptors* sr-57227* ym-31636== antagonists ==antagonists for the receptor (sorted by their respective therapeutic application) include:* antiemetics** as-8112** granisetron** ondansetron** tropisetron* gastroprokinetics** alosetron** batanopride** metoclopramide (high doses)** renzapride** zacopride** m1, the major active metabolite of mosapride* antidepressants** mianserin** mirtazapine** vortioxetine* antipsychotics** clozapine** olanzapine** quetiapine* others** lamotrigine (epilepsy and bipolar disorder)** memantine (alzheimer's disease medication)** menthol** 3-tropanyl indole-3-carboxylate== positive allosteric modulators ==these agents are not agonists at the receptor, but increase the affinity or efficacy of the receptors for an agonist:* 5-chloroindole== discovery ==identification of the 5-ht3 receptor did not take place until 1986 because of a lack of selective pharmacological tool.
==effects==when the receptor is activated to open the ion channel by agonists, the following effects are observed:* cns: nausea and vomiting center in brain stem, anxiety, seizure propensity* pns: neuronal excitation (in autonomic, nociceptive neurons), emesis==agonists==agonists for the receptor include:* 2-methyl-5-ht* ethanol* alpha-methyltryptamine* ibogaine* bufotenin* varenicline* chlorophenylbiguanide* quipazine* rs-56812: potent and selective 5-ht3 partial agonist, 1000x selectivity over other serotonin receptors* sr-57227* ym-31636==antagonists==antagonists for the receptor (sorted by their respective therapeutic application) include:* antiemetics** as-8112** granisetron** ondansetron** tropisetron* gastroprokinetics** alosetron** batanopride** metoclopramide (high doses)** renzapride** zacopride** m1, the major active metabolite of mosapride* antidepressants** mianserin** mirtazapine** vortioxetine* antipsychotics** clozapine** olanzapine** quetiapine* others** lamotrigine (epilepsy and bipolar disorder)** memantine (alzheimer's disease medication)** menthol** 3-tropanyl indole-3-carboxylate==positive allosteric modulators==these agents are not agonists at the receptor, but increase the affinity or efficacy of the receptors for an agonist:* 5-chloroindole==discovery==identification of the 5-ht3 receptor did not take place until 1986 because of a lack of selective pharmacological tool.