Şunu aradınız:: cancer cell death (İngilizce - Maltaca)

this results in cell death.
dan jirriżulta fil-mewt taċ-ċellula.

• mechanisms of pancreas cell death;
• l-impatt tal-fatturi ambjentali fuq ir-regolament u l-progress fuq bażi ġenetika tad-dijabete tat-tip 1;

pixuvri kills cancer cells by binding to dna, resulting in cell death.
pixuvri joqtol iċ-ċelloli tal-kanċer billi jeħel mad-dna, u b’hekk joqtol iċ-ċelloli.

cell death occurs from energy depletion and apoptosis.
il-mewt taċ-ċelluli sseħħ minħabba n-nuqqas ta’ enerġija u apoptożi.

dm1 becomes active once kadcyla enters the cancer cell.
dm1 issir attiva ladarba kadcyla jidħol fiċ-ċellola tal-kanċer.

it also kills cancer cells.
huwa joqtol ukoll iċ-ċelluli tal-kanċer.

this results in bacterial cell death with negligible cell lysis.
din twassal għall-mewt taċ-ċelluli batteriċi fejn ammont insinifikanti biss ta’ ċelluli jiġu distrutti.

enzalutamide treatment decreases the growth of prostate cancer cells and can induce cancer cell death and tumour regression.
il-kura b’enzalutamide tnaqqas it-tkabbir taċ-ċelluli tal-kanċer tal-prostata u tista’ tinduċi l-mewt taċ-ċellula tal-kanċer u r-rigress tat-tumur.

by stopping the cancer cells developing
billi jwaqqaf iċ-ċelluli tal-kanċer milli jiżviluppaw

the cancer cells need this amino acid to
iċ-ċelloli tal-kanċer jeħtieġu dan l-amino aċidu sabiex jikbru u

testosterone can make prostate cancer cells grow.
it-testosteron jista’ jkabbar iċ-ċelloli tal-kanċer tal-prostata.

trifluridine stops the growth of cancer cells.
trifluridine iwaqqaf it-tkabbir taċ-ċelluli tal-kanċer.

inhibition of the 26s proteasome prevents this targeted proteolysis and affects multiple signalling cascades within the cell, ultimately resulting in cancer cell death.
l-inibizzjoni tal-26s proteasome iżomm milli sseħħ din il-proteoliżi u taffettwa l-kaskati ta’ senjalazzjonijiet multipli fiċ-ċellula, li fl-aħħar mill-aħħar iwassal għall-mewt taċ-ċellula tal- kanċer.

antiviral activity is seen at concentrations significantly below those which cause cell death.
l-effett ta’ kontra l-virus jidher anki f’konċentrazzjonijiet ferm anqas li jwasslu għall-mewt ta’ ċelluli

dacogen works by stopping cancer cells from growing.
dacogen jaħdem billi jwaqqaf iċ-ċelluli tal-kanċer milli jikbru.

the lymph nodes can then be checked for cancer cells.
imbagħad, in-nodi limfatiċi jistgħu jiġu ċċekkjati għal ċelloli tal-kanċer.

this means that cancer cells cannot divide and eventually die.
dan ifisser li ċ-ċelloli tal-kanċer ma jistgħux jinqasmu u eventwalment imutu.

it may slow or stop cancer cells from growing, or may kill them.
huwa jista’ jnaqqas jew iwaqqaf iċ-ċelluli tal-kanċer milli jikbru, jew jista’ joqtolhom.

lung cancer cells with mutated egfr proteins are particularly sensitive to afatinib.
iċ-ċelloli tal-kanċer tal-pulmun bi proteini mutati għal egfr huma partikolarment sensittivi għall-afatinib.

by preventing the breakdown of proteins in cancer cells, which are more likely to contain more abnormal proteins, kyprolis causes the death of cancer cells.
bil-prevenzjoni tat-tkissir tal-proteini fiċ-ċelloli tal-kanċer, li għandhom ċans akbar li jkollhom aktar proteini mhux normali, kyprolis joqtol iċ-ċelloli tal-kanċer.