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decreased susceptibility in acinetobacter baumannii has been attributed to the overexpression of the adeabc efflux pump.
nedsat følsomhed over for acinetobacter baumannii er blevet tilskrevet overekspression af adeabc efflukspumpen.
this non-target specific efflux mechanism has also been demonstrated to reduce the in vitro activity of streptogramin a.
det er også vist, at denne non- specifikke efflux mekanisme reducerer in vitro-aktiviteten af streptogramin a.
calcium folinate and folate antagonists share the same membrane transport carrier and compete for transport into cells, stimulating folate antagonist efflux.
calciumfolinat og folatantagonister bruger samme membrantransporter og konkurrerer om transport ind i cellerne, sådan at folatantagonist efflux stimuleres.
decreased susceptibility in both groups has been attributed to the overexpression of the non-specific acrab multi-drug efflux pump.
den nedsatte følsomhed i begge grupper er blevet tilskrevet overekspression af den ikke-specifikke acrab-efflukspumpe til flere lægemidler.
cyp 3a4 inhibition by tipranavir/r and p-gp (an intestinal efflux transporter) inhibition by clarithromycin.
tipranavir/r hæmmer cyp3a4, og clarithromycin hæmmer p-gp (p- glykoprotein, en intestinal efflukstransporter).
one involves mutations in ribosomal protein l3, the other is a non- specific efflux mechanism (abc transporter vgaav).
den ene mekaniske skyldes en mutation i det ribosomale protein l3, mens den anden er en non- specifik efflux- mekanisme (abc transporter vgaav).