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dna polymerase) that are essential for the viruses to reproduce themselves.
tenofovir hu impeditur tan- nukleotide reverse transcriptase; ġeneralment magħruf bħala nrti u jaħdem billi jinterferixxi mal- ħidma normali ta ’ l- enzimi (fl- hiv: reverse transcriptase; fl- epatite b: polymerase tad- dna) li huma essenzjali biex il- viruses jirriproduċu lilhom infushom.
palivizumab does not interfere with reverse transcriptase polymerase chain reaction-based assays.
palivizumab ma jagħmilx effett fuq testijiet li jaħdmu fuq reverse transcriptase polymerase chain reaction.
emtricitabine is a weak inhibitor of mammalian dna polymerase α, β and ε and mitochondrial dna polymerase γ.
emtricitabine hija impeditur dgħajjef tal-polimerat tad-dna mammifera α, β u ε u polimerat mitokondrijali tad-dna γ.
adefovir interferes with the action of a viral enzyme, dna polymerase, which is involved in the formation of viral dna.
adefovir jinterferixxi ma 'l- azzjoni ta' enzima virali, dna polimerażi, li għandha x' taqsam mal- formazzjoni ta 'dna virali.
plasma hcv -rna is measured with a research-based quantitative polymerase chain reaction assay by a central laboratory
hcv-rna fil-plażma jitkejjel b’essej ta’ reazzjoni katina tal-polymerase kwantitattiv ibbażat fuq riċerka minn laboratorju ċentrali.
by competing with the natural substrate deoxyguanosine tp, entecavir-tp functionally inhibits the 3 activities of the viral polymerase:
billi jikkompeti mas- sottostrat naturali deoxyguanosine tp, entecavir tp jaħdem billi ixejjen it- tlett attivitajiet tal- polymerase virali:
simeprevir-resistant variants studied remained susceptible to representative hcv nucleoside and non-nucleoside polymerase inhibitors, and ns5a inhibitors.
varjanti reżistenti għal simeprevir studjati baqgħu suxxetibbli għal impedituri ta’ polymerase nukleosidi u mhux nukleosidi rappreżentattivi ta’ hcv, u impedituri ta’ ns5a.
two novel conserved site mutations were identified in the hbv polymerase gene (rtn236t and rta181v), which conferred clinical resistance to adefovir dipivoxil.
Żewġ mutazzjonijiet ġodda (rtn236t u rta181v) tal-lok konservat kienu identifikati fil-ġene tal-polimerase ta’ hbv, li offrewiet reżistenza klinika għal adefovir dipivoxil.
olaparib is a type of cancer medicine called a parp (poly [adenosine diphosphate-ribose] polymerase) inhibitor.
olaparib huwa tip ta’ mediċina kontra l- kanċer imsejħa inibitur parp (poly [adenosine diphosphate-ribose] polymerase).
telbivudine-5'-triphosphate inhibits hbv dna polymerase (reverse transcriptase) by competing with the natural substrate, thymidine 5'-triphosphate.
telbivudine- 5 ’ - triphosphate jinibixxi hbv dna polymerase (reverse transcriptase) billi jikkompeti mas- sustrat naturali, thymidine- 5 ’ - triphosphate.
abacavir and lamivudine triphosphates show significantly less affinity for host cell dna polymerases.
abacavir u lamivudine triphosphates juru affinita’ sostanzjalment inqas għal polymerases tad-dna taċ-ċellula reċipjenta.