검색어: polymerase (영어 - 몰타어)

polymerase chain reaction
reazzjoni katina tal-polimerażi

nucleotide analogue polymerase inhibitor
impeditur ta’ polymerase analogu nukleotide (nucleotide analogu polymerase inhibitor)

nucleoside analogue dna polymerase inhibitors
impedituri nucleoside analogue dna polymerase

rna polymerases nor an inhibitor of mitochondrial rna polymerase.
rna polymerases, u lanqas mhu inibitur ta’ rna polymerase mitokondrijali.

the entecavir-tp ki for hbv dna polymerase is 0.0012 μm.
il-ki ta’ entecavir-tp għal polymerase tad-dna ta’ hbv huwa

lamivudine-tp acts as a substrate for the hbv viral polymerase.
lamivudine-tp jaħdem bħala substrat għall- polymerase virali tal-hbv.

fidaxomicin is bactericidal and inhibits rna synthesis by bacterial rna polymerase.
fidaxomicin huwa batteriċida u jimpedixxi s-sintesi tal-rna minn rna polymerase batterjali.

dna polymerase) that are essential for the viruses to reproduce themselves.
tenofovir hu impeditur tan- nukleotide reverse transcriptase; ġeneralment magħruf bħala nrti u jaħdem billi jinterferixxi mal- ħidma normali ta ’ l- enzimi (fl- hiv: reverse transcriptase; fl- epatite b: polymerase tad- dna) li huma essenzjali biex il- viruses jirriproduċu lilhom infushom.

specific mutations of rna polymerase are associated with reduced susceptibility to fidaxomicin.
mutazzjonijiet speċifiċi ta’ rna polymerase huma assoċjati ma’ tnaqqis fis-suxxettibilità għal fidaxomicin.

telbivudine is a synthetic thymidine nucleoside analogue with activity against hbv dna polymerase.
telbivudine huwa analogu ta’ thymidine nucleoside sintetiku attiv kontra hbv dna polymerase.

palivizumab does not interfere with reverse transcriptase polymerase chain reaction-based assays.
palivizumab ma jagħmilx effett fuq testijiet li jaħdmu fuq reverse transcriptase polymerase chain reaction.

emtricitabine is a weak inhibitor of mammalian dna polymerase α, β and ε and mitochondrial dna polymerase γ.
emtricitabine hija impeditur dgħajjef tal-polimerat tad-dna mammifera α, β u ε u polimerat mitokondrijali tad-dna γ.

adefovir interferes with the action of a viral enzyme, dna polymerase, which is involved in the formation of viral dna.
adefovir jinterferixxi ma 'l- azzjoni ta' enzima virali, dna polimerażi, li għandha x' taqsam mal- formazzjoni ta 'dna virali.

plasma hcv -rna is measured with a research-based quantitative polymerase chain reaction assay by a central laboratory
hcv-rna fil-plażma jitkejjel b’essej ta’ reazzjoni katina tal-polymerase kwantitattiv ibbażat fuq riċerka minn laboratorju ċentrali.

by competing with the natural substrate deoxyguanosine tp, entecavir-tp functionally inhibits the 3 activities of the viral polymerase:
billi jikkompeti mas- sottostrat naturali deoxyguanosine tp, entecavir tp jaħdem billi ixejjen it- tlett attivitajiet tal- polymerase virali:

simeprevir-resistant variants studied remained susceptible to representative hcv nucleoside and non-nucleoside polymerase inhibitors, and ns5a inhibitors.
varjanti reżistenti għal simeprevir studjati baqgħu suxxetibbli għal impedituri ta’ polymerase nukleosidi u mhux nukleosidi rappreżentattivi ta’ hcv, u impedituri ta’ ns5a.

two novel conserved site mutations were identified in the hbv polymerase gene (rtn236t and rta181v), which conferred clinical resistance to adefovir dipivoxil.
Żewġ mutazzjonijiet ġodda (rtn236t u rta181v) tal-lok konservat kienu identifikati fil-ġene tal-polimerase ta’ hbv, li offrewiet reżistenza klinika għal adefovir dipivoxil.

olaparib is a type of cancer medicine called a parp (poly [adenosine diphosphate-ribose] polymerase) inhibitor.
olaparib huwa tip ta’ mediċina kontra l- kanċer imsejħa inibitur parp (poly [adenosine diphosphate-ribose] polymerase).

telbivudine-5'-triphosphate inhibits hbv dna polymerase (reverse transcriptase) by competing with the natural substrate, thymidine 5'-triphosphate.
telbivudine- 5 ’ - triphosphate jinibixxi hbv dna polymerase (reverse transcriptase) billi jikkompeti mas- sustrat naturali, thymidine- 5 ’ - triphosphate.

abacavir and lamivudine triphosphates show significantly less affinity for host cell dna polymerases.
abacavir u lamivudine triphosphates juru affinita’ sostanzjalment inqas għal polymerases tad-dna taċ-ċellula reċipjenta.