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the quantity of reduced nicotinamide adenine dinucleotide phosphate (nadph) produced corresponds to that of glucose 6-phosphate and thus to that of glucose.
količina proizvedenega reduciranega nikotinamidadenindinukleotidfosfata (nadph) ustreza količini glukoze 6-fosfat in tako tudi količini glukoze.
determination of citric acid in feedingstuff: enzymatic determination of citric content-nadh (reduced form of nicotinamide adenine dinucleotide) spectrometric method.
določanje citronske kisline v krmi: spektrometrična metoda encimskega določanja vsebnosti citronske kisline nadh (reducirana oblika nikotinamid adenin dinukleotida).
l-malic acid (l-malate) is oxidized by nicotinamide adenine dinucleotide (nad) to oxaloacetate in a reaction catalysed by l-malate dehydrogenase (l-mdh).
l-jabolčna kislina (l-malat) se z nikotinamid adenin dinukleotidom (nad) oksidira v oksaloacetat v reakciji, ki jo katalizira l-malat dehidrogenaza (l-mdh).
in the presence of d-malate dehydrogenase decarboxyl (d-mdh), d-malic acid (d-malate) is oxidized by nicotinamide adenine dinucleotide (nad) to oxaloacetate.
v navzočnosti d-malat dehidrogenaze dekarboksila (d-mdh) d-jabolčna kislina (d-malat) oksidira z nikotinamid adenin dinukleotidom (nad) v oksaloacetat.
the glucose 6-phosphate is first oxidized to gluconate 6-phosphate by nicotinamide adenine dinucleotide phosphate (nadp) in the presence of the enzyme glucose 6-phosphate dehydrogenase (g6pdh).
glukoza-6-fosfat najprej oksidira v glukonat-6-fosfat z nikotinamidadenindinukleotidfosfatom (nadp) v navzočnosti encima glukoza 6-fosfat dehidrogenaza (g6pdh).
this buffer solution can be kept for at least four weeks at 4 °c.3.5.nadp (nicotinamide adenine dinucleotide phosphate, disodium salt) solution: dissolve 60 mg of nadp in 6 ml of water.
pufersko raztopino lahko hranimo najmanj štiri tedne pri temperaturi 4°c. 3.5 raztopina nadp (nikotinamid adenin dinukleotid fosfat, di-natrijeva sol): 60 mg nadp raztopimo v 6 ml vode.
if you are allergic to methylthioninium chloride or other thiazine dyes if your body does not produce enough of the enzyme g6pd (glucose-6-phosphate dehydrogenase) if your body does not produce enough of the enzyme nadph (nicotinamide adenine dinucleotide phosphate) reductase
če ste alergični na metiltioninijev klorid ali druga tiazinska barvila če vaše telo ne proizvaja dovolj encima g6pd (glukoza-6-fosfat-dehidrogenaza) če vaše telo ne proizvaja dovolj encima nadph (nikotinamid adenin dinukleotid fosfat) reduktaze
the mechanism of action of cladribine is attributed to the incorporation of cdatp into dna strands: the synthesis of new dna in dividing cells is blocked and the dna repair mechanism is inhibited resulting in an accumulation of dna strand breaks and a decrease of nad (nicotinamide adenine dinucleotide) and atp concentration even in resting cells.
mehanizem delovanja kladribina pripisujemo vgrajevanju cdatp v verige dna: tvorba nove dna v delečih se celicah je onemogočena, zavrt je tudi mehanizem popravljanja dna, kar povzroči kopičenje prelomov verige dna in znižanje koncentracij nad (nikotinamid adenin dinukleotid) in atp tudi v mirujočih celicah.
total lactic acid (l-lactate and d-lactate) is oxidized by nicotinamide adenine dinucleotide (nad) to pyruvate in a reaction catalysed by l-lactate dehydrogenase (l-ldh) and d-lactate dehydrogenase (d-ldh).
skupna mlečna kislina (l-laktat in d-laktat) zaradi nikotinamid adenin dinukleotida (nad) pri reakciji, ki jo katalizirata l-laktat dehidrogenaza (l-ldh) in d-laktat dehidrogenaza (d-ldh), oksidira v piruvat.
when receiving orencia, patients that require blood glucose monitoring should be advised to consider methods that do not react with maltose, such as those based on glucose dehydrogenase nicotine adenine dinucleotide (gdh-nad), glucose oxidase, or glucose hexokinase test methods.
bolnikom, ki potrebujejo merjenje koncentracije krvnega sladkorja, je treba svetovati, da naj med zdravljenjem z zdravilom orencia uporabljajo teste, ki ne reagirajo z maltozo, npr. testi na osnovi glukoza dehidrogenaza nikotin adenin dinukleotidne (gdh-nad), glukoza oksidazne ali glukoza heksokinazne metode.
the main metabolic pathway for tegafur is through conversion to 5-fu via cyp2a6 in the liver, whereas gimeracil was stable in human liver homogenate (s9 fraction) with adenosine 3’-phosphate 5'-phosphosulfphate lithium salt (paps; a co-factor for sulfotransferase) or nicotinamide adenine dinucleotide phosphate (nadph).
glavna presnovna pot za tegafur poteka prek pretvorbe v 5-fu z encimom cyp2a6 v jetrih, medtem ko je gimeracil stabilen v homogenatu človeških jeter (frakcija s9) z adenozin 3’-fosfat 5'-fosfosulfat litijevo soljo (paps; kofaktor za sulfotransferazo) ali nikotinamid adenin dinukleotid fosfatom (nadph).
in the presence of malate dehydrogenase (mdh) and lactate dehydrogenase (ldh), the oxaloacetate and its decarboxylation derivative, pyruvate, are reduced to l-malate and l-lactate by reduced nicotinamide adenine dinucleotide (nadh):oxaloacetate + nadh + h+ l-malate + nad+pyruvate + nadh + h+ l-lactate + nad+ the amount of nadh oxidized to nad+ in these reactions is proportional to the amount of citrate present.
v navzočnosti malat dehidrogenaze (mdh) in laktat dehidrogenaze (ldh) se oksaloacetat in njegov dekarboksilacijski derivat, piruvat, reducirata z reducirano obliko nikotinamid adenin dinukleotida (nadh) v l-malat in l-laktat: oksaloacetat + nadh + h+ l-malat + nad+ piruvat + nadh + h+ l-laktat + nad+ pri teh reakcijah je količina nadh, ki se oksidira v nad+, sorazmerna s količino navzočega citrata.