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reproductive tract infections
infektioner i kønsvejene
Last Update: 2017-04-26
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repr: reproductive tract infection
repr: infektion af forplantningsorganerne
Last Update: 2014-11-21
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findings observed in the male reproductive tract included testicular pachytene spermatocyte degeneration in rats given
fund i reproduktionsvejene hos hanrotter er degenerering af spermatocytter i testis ved doser på ≥50 mg/kg/dag i 28 dage (ca.
other infections of the male or female reproductive tract must meet at least one of the following criteria:
andre infektioner af de mandlige eller kvindelige forplantningsorganer skal opfylde mindst et af følgende kriterier:
particular attention should be paid to the reproductive tract which should be examined for signs of altered development.
særlig opmærksomhed skal udvises over for reproduktionssystemet, som skal undersøges for tegn på forandringer i udviklingen.
significant fab binding was also observed in the male reproductive tract (epididymis, sperm, seminal vesicle) and the skin.
betydende fab-binding blev også observeret i de mandlige forplantningsorganer (epididymis, sperma, vesicula seminalis) og huden.
pyelonephritis, urinary tract infection reproductive system and breast disorders
pyelonefritis, urinvejsinfektion det reproduktive system og mammae usædvanlig:vaginitis
consistent with findings for other beta2 agonists, in lifetime inhalation studies vilanterol trifenatate caused proliferative effects in the female rat and mouse reproductive tract and rat pituitary gland.
i overensstemmelse med resultater med andre beta2-agonister viste livstidsstudier med inhaleret vilanteroltrifenatat proliferative effekter i reproduktionskanalen hos hunrotter og hunmus og i hypofysen hos rotter.
findings observed in the female reproductive tract included single-cell necrosis of ovarian follicles of a rat given 500 mg/kg/day for 3 days.
fund i reproduktionsvejene hos hunrotter er enkeltcellenekrose i ovariefolikler ved doser på 500 mg/kg/dag for 3 dage.
consistent with findings for other beta2-adrenergic agonists, in lifetime inhalation studies, vilanterol trifenatate caused proliferative effects in the female rat and mouse reproductive tract and in the rat pituitary gland.
i overensstemmelse med resultater med andre beta2-agonister viste livstidsstudier med inhaleret vilanteroltrifenatat proliferative virkninger i reproduktionskanalen hos hunrotter og hunmus og i hypofysen hos rotter.
inflammation of the moist lining of mouth and gut, lungs and air passages, reproductive, and urinary tracts,
inflammation (betændelseslignende reaktion) i slimhinder i mund og tarme, lunger og luftveje, kønsdele og urinveje,
in rat and dog repeat-dose toxicity studies up to 6 months duration, target organs for toxicity were gi tract, bone marrow, lymphoid tissues, kidney, adrenal and reproductive tract tissues.
hos rotte og hund i gentaget-dosis toksicitetsstudier op til 6 måneders varighed, var målorganer for toksicitet mave-tarmkanalen, lymfevæv, nyre-, binyre- og reproduktionsvejs-væv.
male fertility was not investigated in the reproductive and developmental toxicity studies, however there were no findings in the repeat-dose toxicity studies to indicate any vaccine-related changes to the tissues of the male reproductive tract.
fertilitet hos hanner blev ikke undersøgt i studierne af reproduktions- og udviklingstoksicitet, men studier af toksicitet efter gentagne doser indikerede ikke vaccine-relaterede ændringer i vævet i forplantningsorganerne hos mænd.
haematuria, kidney calculus, increased creatinine, urinary tract infection, dysuria, urinary retention reproductive system and breast disorders
hæmaturi, nyresten, øget kreatinin, urinvejsinfektion, dysuri, urinretention det reproduktive system og mammae
one year post-natally, there was an increase in the incidence of tumours in the lung, liver and female reproductive tract of offspring exposed to the highest dose level (420 mg/kg term body weight).
et år efter fødslen var der en øget forekomst af tumorer i lunger, lever og hunlige reproduktionsorganer hos afkommet, som var udsat for de højeste doseringsniveauer (420 mg/kg kropsvægt ved termin).
in a juvenile toxicology study in cd rats, administration of 30 mg/ kg/ day of fluoxetine hydrochloride on postnatal days 21 to 90 resulted in irreversible testicular degeneration and necrosis, epididymal epithelial vacuolation, immaturity and inactivity of the female reproductive tract and decreased fertility.
i et toksikologiforsøg med unge cd- rotter resulterede indgift af fluoxetinhydrochlorid 30 mg/ kg/ dag i den postnatale periode fra dag 21 til 90 i en irreversibel degeneration og nekrose af testiklerne, epitelcelle- vakuolisering af bitestiklerne, umodenhed og inaktivitet i forplantningsorganerne hos hunmus samt mindsket yngleevne.
