Vous avez cherché: ergosterol (Anglais - Danois)

itraconazole inhibits the synthesis of ergosterol.
itraconazol hæmmer syntesen af ergosterol.

without ergosterol, the fungus dies or is prevented from spreading.
uden ergosterol dør svampen eller forhindres i at sprede sig.

without ergosterol, the fungus is killed or prevented from spreading.
uden ergosterol dør svampen eller kan ikke brede sig.

it works by disrupting the formation of ergosterol, an important component of fungal cell membranes.
det virker ved at blokere dannelsen af ergosterol, der er en vigtig komponent i svampenes cellemembran.

it works by preventing the formation of ergosterol, which is an important part of fungal cell walls.
det virker ved at forhindre dannelsen af ergosterol, der er en vigtig komponent i svampecellernes væg.

it works by disrupting the formation of ergosterol, which is an important component of fungal cell membranes.
det virker ved at forstyrre dannelsen af ergosterol, som er en vigtig komponent i svampecellemembranerne.

it works by blocking the production of a compound in the fungus called ergosterol which is an important part of fungal cell membranes.
det virker ved at blokere for dannelsen af ergosterol, der er en vigtig komponent i svampecellernes cellemembran.

miconazole nitrate is an antifungal that works by preventing the formation of ergosterol, which is an important part of fungal cell walls.
miconazolnitrat er et middel mod svampe, som virker ved at forebygge dannelse af ergosterol, der er en vigtig komponent i svampecellernes væg.

its mode of action is inhibition of fungal cytochrome p450-mediated 14-sterol demethylation, an essential step in ergosterol biosynthesis.
virkemåden er en hæmning af svampe cytochrom p450-medieret 14-sterol demethylering, som er et essentielt trin i ergosterol biosyntesen.

posaconazole inhibits the enzyme lanosterol 14α-demethylase (cyp51), which catalyses an essential step in ergosterol biosynthesis.
posaconazol hæmmer enzymet lanosterol 14α-demetylase (cyp51), som katalyserer et essentielt trin i ergosterol biosyntesen.

it works by preventing the formation of ergosterol, which is an important part of fungal 7 westferry circus, canary wharf, london e14 4hb, uk tel.
det virker ved at forhindre dannelsen af ergosterol, som er en vigtig del af svampecellemembranerne.uden ergosterol dræbes svampen, eller den forhindres i at sprede sig.listen over svampe, som vfend virker mod, kan ses i produktresuméet.7 westferry circus, canary wharf, london e14 4hb, uk tel.

the primary mode of action of voriconazole is the inhibition of fungal cytochrome p450-mediated 14 alpha-lanosterol demethylation, an essential step in fungal ergosterol biosynthesis.
voriconazols primære virkningsmåde er hæmning af fungal cytochrom p-450-medieret 14-α-lanosteroldemethylering, der er et vigtigt trin i biosyntesen af fungal ergosterol.

reduced susceptibility to triazole antifungal agents has been associated with mutations in the fungal cyp51a and cyp51b genes coding for the target protein lanosterol 14-alpha-demethylase involved in ergosterol biosynthesis.
nedsat følsomhed over for triazol-antimykotika har været forbundet med mutationer i de fungale cyp51a- og cyp51b-gener, der koder for målproteinet lanosterol 14-alpha-demethylase, som er involveret i ergosterols biosyntese.

it selectively inhibits the early synthesis of ergosterol, which is an essential component of the membrane of yeasts and fungi including malassezia pachydermatis (mic90 of 2 µg/ml).
det hæmmer selektivt den tidlige syntese af ergosterol, som er en vigtig del af membranen i gærsvampe og svampe, blandt andet malassezia pachydermatis (mic90 på 2 µg/ml).

(44-20) 74 18 84 00 fax (44-20) 74 18 84 16 e-mail: mail@emea. europa. eu http://www. emea. europa. eu ©emea 2006 reproduction and/or distribution of this document is authorised for non commercial purposes only provided the emea is acknowledged cell membranes.without ergosterol, the fungus is killed or prevented from spreading.the list of fungi against which vfend is active can be found in the summary of product characteristics.
(44-20) 74 18 84 00 fax (44-20) 74 18 84 16 e-mail: mail@ emea. europa. eu http: //www. emea. europa. eu ©emea 2006 reproduction and/or distribution of this document is authorised for non commercial purposes only provided the emea is acknowledged hvordan blev vfend undersøgt undersøgelsen af vfend til behandling af invasiv aspergillosis havde deltagelse af 277 immunforsvarskompromitterede patienter (deres immunsystem fungerede ikke korrekt) .