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pomanjkanje nadph (nikotinamid adenin dinukleotid fosfat) reduktaze.
deficiency in nadph (nicotinamide adenine dinucleotide phosphate) reductase.

raztopina nadp (nikotinamid adenin dinukleotid fosfat, di-natrijeva sol):
nadp (nicotinamide adenine dinucleotide phosphate, disodium salt) solution:

reducirana oblika nikotinamid adenin dinukleotida
reduced form of nicotinamide adenine dinucleotide

raztopina nikotinamid adenin dinukleotida (nad), približno 47 × 10–3 m:
nicotinamide adenine dinucleotide (nad) solution, approximately 47 × 10 3 m:

raztopina reduciranega nikotinamid adenin dinukleotida (nadh) (približno 6 × 10– 3 m):
reduced nicotinamide adenine dinucleotide (nadh) solution (approximately 6 × 10 3 m):

pufersko raztopino lahko hranimo najmanj štiri tedne pri temperaturi 4°c. 3.5 raztopina nadp (nikotinamid adenin dinukleotid fosfat, di-natrijeva sol): 60 mg nadp raztopimo v 6 ml vode.
this buffer solution can be kept for at least four weeks at 4 °c.3.5.nadp (nicotinamide adenine dinucleotide phosphate, disodium salt) solution: dissolve 60 mg of nadp in 6 ml of water.

določanje citronske kisline v krmi: spektrometrična metoda encimskega določanja vsebnosti citronske kisline nadh (reducirana oblika nikotinamid adenin dinukleotida).
determination of citric acid in feedingstuff: enzymatic determination of citric content-nadh (reduced form of nicotinamide adenine dinucleotide) spectrometric method.

če ste alergični na metiltioninijev klorid ali druga tiazinska barvila če vaše telo ne proizvaja dovolj encima g6pd (glukoza-6-fosfat-dehidrogenaza) če vaše telo ne proizvaja dovolj encima nadph (nikotinamid adenin dinukleotid fosfat) reduktaze
if you are allergic to methylthioninium chloride or other thiazine dyes if your body does not produce enough of the enzyme g6pd (glucose-6-phosphate dehydrogenase) if your body does not produce enough of the enzyme nadph (nicotinamide adenine dinucleotide phosphate) reductase

mehanizem delovanja kladribina pripisujemo vgrajevanju cdatp v verige dna: tvorba nove dna v delečih se celicah je onemogočena, zavrt je tudi mehanizem popravljanja dna, kar povzroči kopičenje prelomov verige dna in znižanje koncentracij nad (nikotinamid adenin dinukleotid) in atp tudi v mirujočih celicah.
the mechanism of action of cladribine is attributed to the incorporation of cdatp into dna strands: the synthesis of new dna in dividing cells is blocked and the dna repair mechanism is inhibited, resulting in an accumulation of dna strand breaks and a decrease of nad (nicotinamide adenine dinucleotide) and atp concentration, even in resting cells.

glavna presnovna pot za tegafur poteka prek pretvorbe v 5-fu z encimom cyp2a6 v jetrih, medtem ko je gimeracil stabilen v homogenatu človeških jeter (frakcija s9) z adenozin 3’-fosfat 5'-fosfosulfat litijevo soljo (paps; kofaktor za sulfotransferazo) ali nikotinamid adenin dinukleotid fosfatom (nadph).
the main metabolic pathway for tegafur is through conversion to 5-fu via cyp2a6 in the liver, whereas gimeracil was stable in human liver homogenate (s9 fraction) with adenosine 3’-phosphate 5'-phosphosulfphate lithium salt (paps; a co-factor for sulfotransferase) or nicotinamide adenine dinucleotide phosphate (nadph).