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dpd
dpd

dpd-inducere
dpd inducers

dihydropyrimidindehydrogenase- (dpd-) mangel
dihydropyrimidine dehydrogenase (dpd) deficiency

dihydropyrimidindehydrogenase- (dpd-) mangel.
dpd deficiency.

5-fu metaboliseres af leverenzymet dpd.
5-fu is metabolised by the liver enzyme dpd.

dihydropyrimidindehydrogenase (dpd) aktiviteten er det hastighedsbestemmende trin.
dihydropyrimidine dehydrogenase (dpd) activity is the rate limiting step.

dpd-metoden (dietyl-p-phenylendiamin)
dpd-method (dietyl-p-phenylenediamene)

kendt dpd-mangel (dihydropyrimidin-dehydrogenase).
known dihydropyrimidine dehydrogenase (dpd) deficiency.

patienter med kendt fuldstændig mangel på dihydropyrimidindehydrogenase (dpd) aktivitet,
in patients with known complete absence of dihydropyrimidine dehydrogenase (dpd) activity (see section 4.4),

efter afhændelsen af nfti-ou ledte man forgæves efter en køber til andelen i dpd
after the divestiture in nfti-ou, a buyer for the shareholding in dpd was sought but could not be found.

patienter med kendt dpd-mangel bør ikke behandles med capecitabin (se pkt.
patients with known dpd deficiency should not be treated with capecitabine (see section 4.3).

dpd deficit kan medføre øget toksicitet af capecitabin (se pkt.4. 3 og 4. 4) .
deficiency of dpd may lead to increased toxicity of capecitabine (see section 4.3 and 4.4) .

dpd-mangel er en sjælden, medfødt tilstand, der almindeligvis ikke er forbundet med helbredsproblemer, medmindre man får bestemte lægemidler.
dpd deficiency is a rare condition present at birth that is not usually associated with health problems unless you receive certain medicines.

5-fu nedbrydes yderligere af enzymet dihydropyrimidindehydrogenase (dpd) til det meget mindre toksiske dihydro-5-fluoruracil (fuh2).
5-fu is further catabolised by the enzyme dihydropyrimidine dehydrogenase (dpd) to the much less toxic dihydro-5-fluorouracil (fuh2).