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intracellular pharmacokinetics
16 farmakokinetiċi intraċellulari

erlotinib potently inhibits the intracellular phosphorylation of egfr.
erlotinib jinibixxi b’mod qawwi l-phosphorylation intraċellulari ta’ egfr.

ciclosporin may also increase the intracellular uptake of dexamethasone.
ciclosporin jista’ jżid ukoll it-teħid intraċellulari ta’ dexamethasone.

intracellular ara-gtp accumulated with repeated administration of nelarabine.
ara- gtp akkumula ġewwa ċ- ċellula wara t- tehid ripetut ta 'nelarabine.

docetaxel achieves high intracellular concentrations with a long cell residence time.
docetaxel jinstab f’livelli għoljin fiċ-ċelluli fejn idum għal tul ta’ żmien.

these intracellular metabolites have not been detected in plasma or urine.
dawn il - metaboliti intraċellulari ma ġewx osservati fil- plażma u fl- awrina.

metformin stimulates intracellular glycogen synthesis by acting on glycogen synthase.
metformin jistimola s-sintesi intraċellolari ta’ glycogen billi jaħdem fuq glycogen synthase.

intracellular tenofovir is subsequently phosphorylated to the active metabolite tenofovir diphosphate.
tenofovir intraċellulari jiġi fosforilat sussegwentement għall-metabolit attiv tenofovir diphosphate.

this reduces intracellular sodium accumulation and consequently decreases intracellular calcium overload.
dan inaqqas l-akkumulazzjoni intraċellolari tas-sodju u konsegwentement inaqqas it-tagħbija żejda ta’ kalċju intracellolari.

< 2.0 is the concentration likely to cause significant inhibition of intracellular growth
< 2.0 hija l- konċentrazzjoni li x'aktarx tikkaġuna impediment sinifikanti ta' tkabbir intraċellulari

the intracellular half life of the triphosphate in hepatocytes is 17-19 hours in vitro.
in- nofs ħajja intraċellulari tat- trifosfat fil- hepatocytes hi ta '17- 19 il siegħa in vitro.

lamivudine may inhibit the intracellular phosphorylation of zalcitabine when the two medicinal products are used concurrently.
lamivudine jista 'jimpedixxi l- phosporylation intraċellulari ta ’ zalcitabine meta ż- żewġ prodotti mediċinali jittieħdu flimkien.

polyglutamated metabolites have an increased intracellular half-life resulting in prolonged drug action in malignant cells.
Ġewwa ċ- ċelluli, il-metaboliti poliglutamati għandhom half-life itwal li jwassal biex l-azzjoni tal- mediċina fiċ- ċelluli malinni ddum aktar.

intracellular ara-gtp concentrations in leukaemic blasts were quantifiable for a prolonged period after nelarabine administration.
il- livelli ġewwa ċ- ċellula ta 'ara- gtp f' ċelluli embrijoniċi tal- lewkemja kienu kkwantifikati għal perjodu twil wara t- tehid ta' nelarabine.

sirolimus inhibits t cell activation induced by most stimuli, by blocking calcium dependent and calcium independent intracellular signal transduction.
11 sirolimus jinibixxi l- attivazzjoni taċ- ċelluli t li s- soltu sseħħ permezz ta ’ ħafna stimuli, billi dan jimblokka t- transduzzjoni ta ’ messaġġi intraċellulari kemm dawk li huma u li mhumiex dependenti mill- kalċju.

concentrations (up to 12x) were achieved in the cells (intracellular), compared to the extracellular concentration.
konċentrazzjonijiet (sa 12x) intlaħqu fiċ-ċelluli (intraċellulari), meta mqabbla mal-konċentrazzjoni barra ċ-ċelluli.

concentrations (up to 12 times) were achieved in the cells (intracellular), compared to the extracellular concentration.
konċentrazzjonijiet (sa 12-il darba iktar) inkisbu fiċ-ċelluli (intraċellulari), meta mqabbel mal- konċentrazzjoni ta’ barra ċ-ċelluli.

sirolimus inhibits t-cell activation induced by most stimuli, by blocking calcium-dependent and calcium-independent intracellular signal transduction.
sirolimus jinibixxi l-attivazzjoni taċ-ċelluli t ikkawżata mill-biċċa l-kbira tal-istimuli, billi dan jimblokka t-transduzzjoni ta’ messaġġi intraċellulari kemm dawk li huma u li mhumiex dipendenti mill-kalċju.

everolimus binds to the intracellular protein fkbp-12, forming a complex that inhibits mtor complex-1 (mtorc1) activity.
everolimus jeħel mal-proteina intraċellulari fkbp-12, u jifforma kumpless li jimpedixxi l- attività ta’ mtor complex-1 (mtorc1).